Buffdoc-
- Thread starter house1
- Start date
The fact that your GGTP is so low is VERY encouraging. Intense workout can elevate SGOT (AST) for sure, and probabaly SGPT (ALT) too.
However, the fact that the SGPT is MORE elevated that the SGOT is a little worrysome. Is your doc a Gastroenterologist? Did you say these had been up before? What about the Hepatitis panel I was asking you about. Sory if I didn't see another response; I've been gone all day.
However, the fact that the SGPT is MORE elevated that the SGOT is a little worrysome. Is your doc a Gastroenterologist? Did you say these had been up before? What about the Hepatitis panel I was asking you about. Sory if I didn't see another response; I've been gone all day.
house1
Community Veteran
no hepatitis buff .
last august they were slightly elevated but not bad at all.
Is your doc a Gastroenterologist? no and what do they specialize in?
buff, i had a camera shoved in my ass lol and down my throat last september
i have acid reflux which caused berrets disease. i have to go every year now to get that looked at.
he wants to send me back if things do not come down. i will be going for blood work again in august.
last august they were slightly elevated but not bad at all.
Is your doc a Gastroenterologist? no and what do they specialize in?
buff, i had a camera shoved in my ass lol and down my throat last september
i have acid reflux which caused berrets disease. i have to go every year now to get that looked at.
he wants to send me back if things do not come down. i will be going for blood work again in august.
house1 said:
Hey bro,
Gastroenterology is the specialty of diseases of the GI tract, including the liver and gallbladder. The guy that reamed ya with the scopes was probably a GE; they do most of that stuff.
If no hepatitis, and minimal elevations, you may be ok (as long as they've looked for the rare stuff like hemochromatosis, Wilson's disease, autoimmune hepatitis, etc). I'd be VERY interested to see your levels after not working out for a week (although I can't think of a time they could get ME w/ a week off!)
hhajdo
Community Veteran
As buffdoc said, it might be a good idea to get your levels checked again after not working out for a week, since ALT & AST could be elevated due to intense resistance training (muscle damage)...
Evaluation of aminotransferase elevations in a bodybuilder using anabolic steroids: hepatitis or rhabdomyolysis?
Pertusi R, Dickerman RD, McConathy WJ.
Department of Medicine, University of North Texas Health Science Center, 3500 Camp Bowie Blvd, Fort Worth, TX 76107-2699, USA.
The use of anabolic steroids among competitive athletes, particularly bodybuilders, is widespread. Numerous reports have noted "hepatic" dysfunction secondary to anabolic steroid use based on elevated serum aminotransferase levels. The authors' objective was to assess whether primary care physicians accurately distinguish between anabolic steroid-induced hepatotoxicity and serum aminotransferase elevations that are secondary to acute rhabdomyolysis resulting from intense resistance training. Surveys were sent to physicians listed as practicing family medicine or sports medicine in the yellow pages of seven metropolitan areas. Physicians were asked to provide a differential diagnosis for a 28-year-old, anabolic steroid-using male bodybuilder with an abnormal serum chemistry profile. The blood chemistries showed elevated aspartate aminotransferase (AST), alanine aminotransferase (ALT), and creatine kinase (CK) levels, and normal gamma-glutamyltransferase (GGT) levels. In the physician survey (n = 84 responses), 56% failed to mention muscle damage or muscle disease as a potential diagnosis, despite the markedly elevated CK level of the patient. Sixty-three percent indicated liver disease as their primary diagnosis despite normal GGT levels. Prior reports of anabolic steroid-induced hepatotoxicity that were based on aminotransferase elevations may have overstated the role of anabolic steroids. Correspondingly, the medical community may have been led to emphasize anabolic steroid-induced hepatotoxicity and disregard muscle damage when interpreting elevated aminotransferase levels. Therefore, when evaluating enzyme elevations in patients who use anabolic steroids, physicians should consider the CK and GGT levels as essential elements in distinguishing muscle damage from liver damage.
Evaluation of aminotransferase elevations in a bodybuilder using anabolic steroids: hepatitis or rhabdomyolysis?
Pertusi R, Dickerman RD, McConathy WJ.
Department of Medicine, University of North Texas Health Science Center, 3500 Camp Bowie Blvd, Fort Worth, TX 76107-2699, USA.
The use of anabolic steroids among competitive athletes, particularly bodybuilders, is widespread. Numerous reports have noted "hepatic" dysfunction secondary to anabolic steroid use based on elevated serum aminotransferase levels. The authors' objective was to assess whether primary care physicians accurately distinguish between anabolic steroid-induced hepatotoxicity and serum aminotransferase elevations that are secondary to acute rhabdomyolysis resulting from intense resistance training. Surveys were sent to physicians listed as practicing family medicine or sports medicine in the yellow pages of seven metropolitan areas. Physicians were asked to provide a differential diagnosis for a 28-year-old, anabolic steroid-using male bodybuilder with an abnormal serum chemistry profile. The blood chemistries showed elevated aspartate aminotransferase (AST), alanine aminotransferase (ALT), and creatine kinase (CK) levels, and normal gamma-glutamyltransferase (GGT) levels. In the physician survey (n = 84 responses), 56% failed to mention muscle damage or muscle disease as a potential diagnosis, despite the markedly elevated CK level of the patient. Sixty-three percent indicated liver disease as their primary diagnosis despite normal GGT levels. Prior reports of anabolic steroid-induced hepatotoxicity that were based on aminotransferase elevations may have overstated the role of anabolic steroids. Correspondingly, the medical community may have been led to emphasize anabolic steroid-induced hepatotoxicity and disregard muscle damage when interpreting elevated aminotransferase levels. Therefore, when evaluating enzyme elevations in patients who use anabolic steroids, physicians should consider the CK and GGT levels as essential elements in distinguishing muscle damage from liver damage.
house1
Community Veteran
buff, these are my levels last august. i was out with a shoulder injury which i had surgery on in sept. so there was about 4 weeks of no lifting before these were done. my tests 5 months before those were high aswell. i lift heavy all the time can that be causing the high values
sgot 39 range 10-35
sgpt 45 range 8-40
ggtp 20 range 7-51
there not bad
sgot 39 range 10-35
sgpt 45 range 8-40
ggtp 20 range 7-51
there not bad
