How long does Human Chorionic Gonadotropin (HCG) stay effective


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How long does HCG stay effective

After mixing the two together does any one know how long Human Chorionic Gonadotropin (HCG) will stay effecitve? The reason I ask is because on a manufacturers website for a particular brand of Human Chorionic Gonadotropin (HCG) it says to use within 24 hours or so after mixing the powder and water together. I'm hoping that it stays good (PROVIDED ITS REFRIDGERATED) for close to a year or more. :confused: :confused:
All the bottles I ever had say use within 30 days of refrigeration. I would think it would be good a bit longer than that, but, I have nothing to back that up with....
A note on HCG...

I read a study that said analgesics (tylenol, advil etc) reduce the effect of Human Chorionic Gonadotropin (HCG) substantially.
gorilla_boy said:
A note on HCG...

I read a study that said analgesics (tylenol, advil etc) reduce the effect of Human Chorionic Gonadotropin (HCG) substantially.

Interesting, never heard that....
Thanks guys,
gorillaboy didnt kn0w that, I feel ya on elite but your just helpin a bro out, I wont go there too much anymore this is my new home:D
Aspirin inhibits androgen response to Human Chorionic Gonadotropin ( Human Chorionic Gonadotropin (HCG) )

Aspirin inhibits androgen response to chorionic gonadotropin in man. Conte, Domenico, Francesco Romanelli, Silvia Fillo, Laura Guidetti, Aldo Isidori, Francesco Franceschi, Maurizio Latini, and Luigi Di Luigi. Division of Andrology, Department of Medical Pathophysiology, University _La Sapienza_, Rome, Italy; * Laboratory of Endocrinological Research, University Institute of Motor Sciences, Rome, Italy; and **Department of Orthopedics, Libera Universit[grave]a, Campus Biomedico, Rome, Italy
APStracts 6:0189E, 1999.

Eicosanoids play an important role in the regulation of the hypothalamic-pituitary axis; less clear is their role in testicular steroidogenesis. To evaluate the involvement of cycloxygenase metabolites, such as prostaglandins, in the regulation of human testicular steroidogenesis, we examined the effects of a prostaglandin-blocker, aspirin, on plasma testosterone, pregnenolone, progesterone, 17OH-progesterone, androstenedione, dehydroepiandrosterone and 17[beta](estradiol response to hCG in normal male volunteers in a placebo controlled, single blinded study. To test the efficacy of aspirin, seminal prostaglandin E2 levels were also determined. hCG stimulation increased peripheral levels of testosterone, 17OH-progesterone, androstenedione, dehydroepiandrosterone and 17[beta](estradiol, without affecting circulating pregnenolone and progesterone values. Aspirin significantly lowered seminal prostaglandin E2 levels whereas it did not modify steroid concentrations not exposed to exogenous hCG. Moreover, the drug significantly reduced the response of testosterone, 17OH-progesterone, androstenedione and dehydroepiandrosterone to hCG, assessed by the mean integrated area under the curve, whereas it did not influence 17[beta](estradiol response. In conclusion, aspirin treatment inhibits androgen response to chorionic gonadotropin stimulation in normal humans. The action of aspirin is probably mediated via an effective arachidonate cycloxygenase block.