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Acetyl-L-Carnitine Increases Androgen Receptors
Renowned muscle researcher Dr. Bill Kraemer has published a handful of excellent studies showing that the acetylated form of L-Carnitine increases androgen receptor content following whole-body resistance training workouts.(1)
Kraemer showed that 21 days of supplementation with acetyl-L-carnitine led to a significant increase in the androgen-receptor content of the vastus lateralis muscle in the thigh. (The vastus lateralis is the traditional spot from which to take muscle biopsies and if the number of androgen receptors increased there, they increased in all muscles.)
Additionally, Kraemer noted that the athletes' blood levels of Testosterone plummeted after a workout. While that initially sounds like a bad thing, it's not. Instead, it shows that something good happened.
Since the "roads" (blood vessels) had less Testosterone flowing through them, it meant that Testosterone had pulled off the road and "parked" in the new Testosterone receptors found on the acetyl-L-carnitine user's muscle.
Since there were more parking spots for Testosterone to park, it leads to more protein synthesis, which literally means more muscle.
Furthermore, the temporarily low level of free Testosterone in the blood stream tells the brain (the pituitary-gonadal axis), through negative feedback, that it needs to start producing even more Testosterone.
Does anyone want to testify in favor of this?
Renowned muscle researcher Dr. Bill Kraemer has published a handful of excellent studies showing that the acetylated form of L-Carnitine increases androgen receptor content following whole-body resistance training workouts.(1)
Kraemer showed that 21 days of supplementation with acetyl-L-carnitine led to a significant increase in the androgen-receptor content of the vastus lateralis muscle in the thigh. (The vastus lateralis is the traditional spot from which to take muscle biopsies and if the number of androgen receptors increased there, they increased in all muscles.)
Additionally, Kraemer noted that the athletes' blood levels of Testosterone plummeted after a workout. While that initially sounds like a bad thing, it's not. Instead, it shows that something good happened.
Since the "roads" (blood vessels) had less Testosterone flowing through them, it meant that Testosterone had pulled off the road and "parked" in the new Testosterone receptors found on the acetyl-L-carnitine user's muscle.
Since there were more parking spots for Testosterone to park, it leads to more protein synthesis, which literally means more muscle.
Furthermore, the temporarily low level of free Testosterone in the blood stream tells the brain (the pituitary-gonadal axis), through negative feedback, that it needs to start producing even more Testosterone.
Does anyone want to testify in favor of this?
