It doesnt stimulate test production as much as tamox, torem or clomid but it does bind more strongly to the e receptor in breast tissue more strongly than they do. This makes it better suited for Gyno treatment than PCT although in a gyno situation I see no reason why it could not successfully be worked into a pct protocol.
It doesnt stimulate test production as much as tamox, torem or clomid but it does bind more strongly to the e receptor in breast tissue more strongly than they do. This makes it better suited for Gyno treatment than PCT although in a gyno situation I see no reason why it could not successfully be worked into a pct protocol.
Not trying to hi-jack guys, but I do have a question that is on topic. Suppose you start to develop gyno while on cycle. What would the best treatment option be?
Then up your AI dose... You have no reason to cycle without one.
Or, up your AI dose as it is an indication your e2 is too high and use a SERM (Raloxifene would be best, however Nolva or Toremifene will also do the trick, Clomid I wouldn't waste your time with for breast tissue antagonism).
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