I agree completely with Kaz here. You will not get a kick start by frontloading, the metabolic rate of the compound is determined by the ester mainly. You could front load at 1200mg but it won't kick in any faster, it'll just kick in harder when it kicks in, which again is the same time. I'd just drop the EQ bro.
D you have experience interpreting blood work? Do you plan on even getting blood work? Do you know EQ IS ONE OF THE WORST compounds in terms of how it affects your RBC's and HCT levels causing polycythemia, clogged arteries and veins, blood clots, etc? Do you know how to lower RBC'S and HCT if necessary which will be pretty much a sure thing? If you don't know these things you really have no business running EQ and as mentioned before, stick to test alone.
Also EQ
IN MINIMAL DOSES CAN HAVE A STRENGTHENING AFFECT ON TENDONS. There is nothing minimal about a 1200mg frontload and even a 500mg/wk dose. Even testosterone have an adverse affect on bones and tendon strength with anything higher than a testosterone replacement therapy (TRT) dose. I don't know where you got your information from but I can provide you with the relavent studies. And if you try to provide evidence to the contrary please let it be credible evidence, not a steroid.com article or something off Wikipedia. I'm talking scientific PEER REVIEWED studies. Like I said, if you're interested in good long reading ill provide links but I expect the same from you, credible links.
I never said HCG was only for testicular atrophy. I know cholesterol is a base for hormonal production but what you're failing to understand is
Youre taking exogenous testosterone. Not only will this cause suppression of natural testosterone with or without HCG but you're getting supra physiological levels by injecting it, the 2.5-11mg/day you make naturally totals up to 77mg/wk tops. Is this really what your concerned about? An extra 77mg/wk. the other thing you're missing is that testosterone CAN be made by cholesterol and progesterone BUT ITS NOT THE PREFERRED PATHWAY NOR IS IT THE ONLY THING THAT CAN BE MADE. The preffered method for testosterone production is cholesterol to pregnenolone to 17-hydropregnenolone to DHEA which them makes testosterone from androstenadiol or androstenedione. When testosterone is made from progesterone it means the pregnenolone is being "robbed" from making DHEA since DHEA can only be made from pregnenolone and thus the pathway is hydroxyprogesterone to androstenidione to testosterone. The problem with this is since progesterone is being made to estrogen and testosterone, it's ability to make cortiso amd aldosteronel is impaired (these are necessary hormones). The other mistake is to assume ONLY testosterone is being made from cholesterol. In fact cholesterol can make aldosterone, cortisol, estradiol, estrone, DHEA and many other hormones. How do you know which one you're making? You assume it's testosterone but in reality it is many hormones including estrogen.
View attachment 551834
The Endocrine Journey: Steroidal Hormones & Their Pathways
88.05.04: The Effects that the Following Hormones (Testosterone, Estrogen, and Progesterone) Have on the Human Body
And finally the last thing you assume is how it affects cholesterol levels. Testosterone usually influences HDL more than LDL and its LDL that's used for testosterone and other hormonal production, so how does increasing test production with HCG prevent accumulation? It's your LDL levels that create test and other hormones but usually HDL that gets affected by testosterone use??? Doesn't make sense. Now LDL can also be affected but testosterone Can lowers LDL, again how's it being accumulated? And finally if your worried about cholesterol on cycle so much, you want higher estrogen levels than normal because estrogens have a favorable impact on cholesterol. This will have a greater effect on cholesterol than trying to use it to make testosterone which is a double assumption bc you don't know if testosterone is being made or another hormone and than you assume it's LDL not HDL.
***8220;important to note that estrogens can have a favorable impact on cholesterol profiles. The aromatization of testosterone to estradiol may, therefore***8221;
Excerpt From: Llewellyn, William. ***8220;Anabolics.***8221; iBooks.
This material may be protected by copyright.
https://itunes.apple.com/us/book/anabolics/id486921298?mt=11