NickalausA87
New member
What is the best way to cycle this stack for best results! First time with the masteron and bit of a rookie with tren
It's all about timing your compounds/esters...is the mast prop?
Since you have a lot of short esters, EOD/E3D would be sufficient...
You are also going to have to become familiarized with injection sites and what's available, proper protocols, what muscle groups to avoid and what groups are more favored...
I would suggest getting a smaller gauge needle 25g 1.0 could be utilized on a few sites such as your deltoids and your quad,or VG, 1.5 25g for glutes.. I suggest this due to the fact that you will have a lot of injection and this will assist with irritation and scar tissue... depending on the quality of your compounds and the carrier oils that have been utilized along with the solvent ratio, they should be smooth in a 25.. If not you can heat up the needle with a blowdryer... this will change the viscosity considerably, temporarily making it easier for injection...
I only suggest that protocol because of your pinning and the schedule will be more frequent...
1mL of each compound should be sufficient.. Depending on your goal..
Limit oil concentration in your sites...delt 1.5mL max ( these compounds will have a bite) , glute 2-3 mL, quad 1-2 mL max...VG 1-2mL
How funny, i can get 3ml into pecs delts lats quads and traps. More into glutes. Haven't tried tris and bis but i guess my cruise dosage would work there
It's all about timing your compounds/esters...is the mast prop?
Since you have a lot of short esters, EOD/E3D would be sufficient...
You are also going to have to become familiarized with injection sites and what's available, proper protocols, what muscle groups to avoid and what groups are more favored...
I would suggest getting a smaller gauge needle 25g 1.0 could be utilized on a few sites such as your deltoids and your quad,or VG, 1.5 25g for glutes.. I suggest this due to the fact that you will have a lot of injection and this will assist with irritation and scar tissue... depending on the quality of your compounds and the carrier oils that have been utilized along with the solvent ratio, they should be smooth in a 25.. If not you can heat up the needle with a blowdryer... this will change the viscosity considerably, temporarily making it easier for injection...
I only suggest that protocol because of your pinning and the schedule will be more frequent...
1mL of each compound should be sufficient.. Depending on your goal..
Limit oil concentration in your sites...delt 1.5mL max ( these compounds will have a bite) , glute 2-3 mL, quad 1-2 mL max...VG 1-2mL
Then why don't you suggest this protocol to him? 3mls,straight to his pecs with short esters,like a boss..lol..anyhow, we don't know his body composition and his size, especially considering the compounds that he has at hand and the site irritation those estern posses.. if you wish to put 3ml in your pecs that's fine, that's something I would never do... compartment syndrome cellulitis is not a joke it could happen to anyone at any given time, your lymphatic system can be rather sassy.. the last thing I think this guy wants is to be knotted up!![]()
Then why don't you suggest this protocol to him? 3mls,straight to his pecs with short esters,like a boss..lol..anyhow, we don't know his body composition and his size, especially considering the compounds that he has at hand and the site irritation those estern posses.. if you wish to put 3ml in your pecs that's fine, that's something I would never do... compartment syndrome cellulitis is not a joke it could happen to anyone at any given time, your lymphatic system can be rather sassy.. the last thing I think this guy wants is to be knotted up!![]()
I would not be concerned about Compartment Syndrome from injection cellulitis. That is extremely rare. Extremely! Compartment Syndrome is most often associated with Crush Injuries.
I am not saying it isn't possible. I am saying it is rare. Compartment Syndrome should not be one's first thought. Here is a medical info for consideration.
Non-Necrotizing Streptococcal Cellulitis as a Cause of Acute, Atraumatic Compartment Syndrome of the Foot: A Case Report. - PubMed - NCBI
Non-Necrotizing Streptococcal Cellulitis as a Cause of Acute, Atraumatic Compartment Syndrome of the Foot: A Case Report.
Toney J1, Donovan S2, Adelman V2, Adelman R3.
Author information
Abstract
Acute compartment syndrome is widely accepted as a surgical emergency. Most cases of acute compartment syndrome occur after high-energy trauma, especially crush injuries. We present a unique case of acute, atraumatic compartment syndrome of the foot associated with infectious cellulitis. A 53-year-old male, with a medical history significant for human immunodeficiency virus, presented to the emergency department secondary to an insidious onset of intense foot pain, swelling, and an inability to bear weight on the affected extremity. He had no history of recent trauma. He was admitted to the hospital because of a suspected infection and subsequently was given intravenous antibiotics. During the admission, he developed a severe infection, and blood cultures demonstrated growth of group A streptococcus. No abscess or hematoma was identified on magnetic resonance imaging or during exploratory surgery. The findings from intraoperative cultures were negative. Despite proper medical care for his infection, the lower extremity pain worsened; therefore, compartmental pressures were obtained at the bedside. Multiple compartment pressures were measured and were >40 mm Hg. Compartment syndrome was diagnosed, and the patient was taken to the operating room for emergent fasciotomies. Surgical release of the medial, lateral, interosseous, and adductor compartments revealed copious amounts of serosanguinous drainage. Again, no definitive hematoma or purulence was identified. The patient's symptoms resolved after the fasciotomies, and he healed uneventfully. Our case highlights the need to consider acute compartment syndrome in the differential diagnosis for pain out of proportion to the clinical situation, even when a traditional etiology is absent.
This is baseless, and irrelevant to the topic. Yes this can happen with an injury, but why are you utilizing this to support anything beats me? I mentioned cellulitis through the means of injections, and you post a foot injury by force? lol come on mega.
Its more common vs RARE in this lifestyle. Ive had it TWICE, and Ive seen it happen to others..The ER doctor mention to me that he has seen it in many cases with young men using AAS, and on my 2nd stint at a different hosp the operating Dr also questions my usage and after we discussed some things (he was trying to piece things together) he also mentioned his large base of patients with my similar instance through means of AAS usaged.. How many times have we read a post about injection swelling and someone seeking advise,and later they end up being prescribed Bactrim (I was prescribed this as well for my situation,even though it appeared to be dermal/subq,when it fact the Dr was aware it was IM)..If its pink/red on the outside, you can bet its tan/brown and almost black on the inside..
when people get a bad injection not every case is small immune response/subq tissue or hitting nerves ext,and when they have localized swelling, what do you think is happening? shit can spiral out of control, fast!
If anyone believe the facts I stated prior not true, then Im sure a demitasses would fit their head like a sombrero!
I'll see you on your "Old man with a smashed foot" and raise you a STEROID INJECTION cellulitis article..
Necrotizing myositis of the deltoid following intramuscular injection of anabolic steroid
A 25-year-old male bodybuilder was admitted in septic shock. He had marked limitation of left shoulder movement with non- fluctuant swelling extending from the acromio-clavicular joint to the postero-lateral and anterior deltoid. Lesions consistent with herpes labialis were also noted. A computed tomography scan demonstrated collections of both gas and fluid within the deltoid muscle and numerous pockets of gas within the soft tissues conforming to the fascial planes (Figure 1). On further questioning he admitted to injecting himself with anabolic steroids into the left deltoid region six days prior to admission. He underwent emergency drainage with debridement of large areas of necrotic muscle and a fasciotomy of the upper arm. Gemella morbillorum and Veillonella were isolated from culture of pus. Dialister pneumosintes was grown from blood cultures.
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The strains were identified by 16 s rDNA sequencing. He was treated with benzyl penicillin and clindamycin. Histology showed necrotic skeletal muscle tissue with mixed inflammatory infiltrate. Despite extensive muscle debridement he retained excellent shoulder function. The use of anabolic steroids for performance enhancement amongst young adults is well known. According to a Home Office estimate, there are more than 40 000 users of anabolic steroids in the UK.. As many as 36% of anabolic steroid users are not athletes. The pathogens recovered in the setting of anabolic steroid injection-related abscess include Staphylococcus aureus, Streptococcus, and Pseudomonas. Both solitary and multiple abscesses have been reported. A solitary abscess may result from a ***8216;spot shot***8217;, where injection is used to build a specific muscle group, such as the deltoid or the pectoral muscle.
The common mode of acquisition of infection is thought to be related to needle contamination with skin flora and due to contaminated drug.1 G. morbillorum is part of the normal flora of the respiratory tract and was once classified as a member of the viridans Streptococcus family. Veillonella and Dialister are rare anaerobic pathogens. They are part of the normal oral and gut flora. Systemic infection has been documented following injection use,5 after endoscopic procedures,6 and in immunocompromised patients.7 Herpes viruses are known to activate periodontal bacteria by impairing local defense mechanisms.8 High doses of anabolic steroids with an intact steroid nucleus are immunosuppressive, while steroids with an altered nucleus are known to stimulate the T lymphocytes.9 We are unsure as to the chemical nature of the steroid in this case as it was acquired illicitly. Although the exact mode of infection is uncertain, the triad of anabolic steroid-mediated immunosuppression, reactivation of herpes simplex, and anaerobic bacteremia with seeding of a traumatized area in the deltoid could explain the pathogenesis in this patient.
http://www.ijidonline.com/article/S1201-9712(10)02393-3/fulltext
Necrotizing myositis of the deltoid following intramuscular injection of anabolic steroid
A 25-year-old male bodybuilder was admitted in septic shock. He had marked limitation of left shoulder movement with non- fluctuant swelling extending from the acromio-clavicular joint to the postero-lateral and anterior deltoid. Lesions consistent with herpes labialis were also noted. A computed tomography scan demonstrated collections of both gas and fluid within the deltoid muscle and numerous pockets of gas within the soft tissues conforming to the fascial planes (Figure 1). On further questioning he admitted to injecting himself with anabolic steroids into the left deltoid region six days prior to admission. He underwent emergency drainage with debridement of large areas of necrotic muscle and a fasciotomy of the upper arm. Gemella morbillorum and Veillonella were isolated from culture of pus. Dialister pneumosintes was grown from blood cultures.
![]()
The strains were identified by 16 s rDNA sequencing. He was treated with benzyl penicillin and clindamycin. Histology showed necrotic skeletal muscle tissue with mixed inflammatory infiltrate. Despite extensive muscle debridement he retained excellent shoulder function. The use of anabolic steroids for performance enhancement amongst young adults is well known. According to a Home Office estimate, there are more than 40 000 users of anabolic steroids in the UK.. As many as 36% of anabolic steroid users are not athletes. The pathogens recovered in the setting of anabolic steroid injection-related abscess include Staphylococcus aureus, Streptococcus, and Pseudomonas. Both solitary and multiple abscesses have been reported. A solitary abscess may result from a ***8216;spot shot***8217;, where injection is used to build a specific muscle group, such as the deltoid or the pectoral muscle.
The common mode of acquisition of infection is thought to be related to needle contamination with skin flora and due to contaminated drug.1 G. morbillorum is part of the normal flora of the respiratory tract and was once classified as a member of the viridans Streptococcus family. Veillonella and Dialister are rare anaerobic pathogens. They are part of the normal oral and gut flora. Systemic infection has been documented following injection use,5 after endoscopic procedures,6 and in immunocompromised patients.7 Herpes viruses are known to activate periodontal bacteria by impairing local defense mechanisms.8 High doses of anabolic steroids with an intact steroid nucleus are immunosuppressive, while steroids with an altered nucleus are known to stimulate the T lymphocytes.9 We are unsure as to the chemical nature of the steroid in this case as it was acquired illicitly. Although the exact mode of infection is uncertain, the triad of anabolic steroid-mediated immunosuppression, reactivation of herpes simplex, and anaerobic bacteremia with seeding of a traumatized area in the deltoid could explain the pathogenesis in this patient.
http://www.ijidonline.com/article/S1201-9712(10)02393-3/fulltext
As usual, you are missing the point.
See what I highlighted for you in red and bold. That is the point -- the takeaway message. I tried to make it very simple for you. The rest is acknowledging that cellulitis can be a cause of Compartment Syndrome as you pointed out. But simply because it happened to you twice or to a guy with a foot infection does not make it common or something that most people should worry about. Especially those in good health and injecting steroids. It is NOT common in weight lifting or AAS users unless they get crushed. You keep taking information and twisting it in a way that is dangerous if people listen to you. The forum would be better served if you stuck to repping in my personal opinion.
I get the sense that English may not be your native language...
This is an example of an infection or Sterile Abscess. NOT Compartment Syndrome. I don't think you understand what Compartment Syndrome is.
Necrotizing myositis ----- Is exactly how I got cellulitis COMPARTMENT SYNDROME!
It does not have to be a crushing blow to make tissue swell, in fections also causes tissue to swell..You silly goose..
Obviously the crow is white on your end.. I see a black crow all day!
I dont think YOU have a clue, let alone experienced it,But I did, twice like I said, with 2 major operations and the removal of tissue..and your stand fast on a smashed foot.. You win! It was fun chatting.. enjoy your holiday fine sir..
BTW
There's tons and tons or reads you can find in regards to this, I have 2 saved somewhere, deep..I can pull them up later..Have at it and read about..
What shocks me here Mega, is sometimes in your post your spot on with things, this one brutha, Im disappointed to say the least!
I'll say this one last time.. CS is not only caused by trauma.. READ ABOUT IT! I had it twice..
nuff said..
I have agreed already that it can happen. But I do not agree with you that it is a common cause of Compartment Syndrome. I don't think you understand what I have been writing.