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Strasseroids
Underground steroid information from around the world
by Brock Strasser
Eating Your Liver With a Nice Chianti?
Q: My boyfriend has decided to try steroids for the first time after discussing them with me. While I’m grateful that our relationship is honest and open enough for him to talk to me about this, I’m concerned about his choice of steroids. We both decided that he shouldn’t use any injectable stuff, so please, let’s not even mention this.
My guy is planning on using 25 mg of D-bol for four weeks. Two weeks into this he plans to add 50 mg of something called Anapolan, which I heard was really liver toxic. He plans to stay on this for four weeks and two weeks into the Anapolan he’s planning to add in 30 mg of Winstrol taken orally for four weeks. Is this safe to his liver? I’ve read a lot of horrible things about oral steroid use being associated with liver cancer and cirrhosis. If this isn’t safe, what orals to you suggest instead of these?
A: For the longest time, I’ve been trying to dispel the urban myth that steroids cause liver cancer and liver damage. I’ve repeatedly written and stated that most people, especially if they don’t have any underlying health conditions, can use oral steroids for 10 to 12 weeks per year every year for a decade and not suffer from any lasting ill effects. Here’s a study I found that gives much credence to my "philosophy":
Anabolic steroid-induced hepatotoxicity: Is it overstated?
Clin J Sport Med 1999 Jan;9(1):34-9 (ISSN: 1050-642X)
Dickerman RD; Pertusi RM; Zachariah NY; Dufour DR; McConathy WJ
The Department of Biomedical Science, University of North Texas Health Science Center, Fort Worth 76107-2699, USA.
OBJECTIVE: There have been numerous reports of hepatic dysfunction secondary to anabolic steroid use based on elevated levels of serum aminotransferases. This study was conducted to distinguish between serum aminotransaminase elevations secondary to intense resistance training and anabolic steroid-induced hepatotoxicity in elite bodybuilders.
DESIGN: This was a case-control study of serum chemistry profiles from bodybuilders using and not using anabolic steroids with comparisons to a cohort of medical students and patients with hepatitis.
PARTICIPANTS: The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and nonexercising medical students (592) were used as controls.
MAIN OUTCOME MEASURES: The focus in blood chemistry profiles was aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels.
RESULTS: In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT.
CONCLUSION: Prior reports of anabolic steroid-induced hepatotoxicity based on elevated aminotransferase levels may have been overstated, because no exercising subjects, including steroid users, demonstrated hepatic dysfunction based on GGT levels. Such reports may have misled the medical community to emphasize steroid-induced hepatotoxicity when interpreting elevated aminotransferase levels and disregard muscle damage. For these reasons, when evaluating hepatic function in cases of anabolic steroid therapy or abuse, CK and GGT levels should be considered in addition to ALT and AST levels as essential elements of the assessment.
Now I’m sure that if someone "lived" on oral steroids (by oral, I’m referring to those that are alpha alkylated at carbon 17) week in and week out for years, he’d end up with serious health issues. But based on what you’ve written, I’d guess that your boyfriend (without knowing anything else about him) will handle this quite well and if he eats and trains correctly, he should gain upwards of 10 to12 pounds of solid muscle from this cycle.
I’d suggest he consider adding in an anti-aromatase (Arimidex) or anti-estrogen (Nolvadex, Clomid) during the time he’s on D-bol and for one week after.
He should also expect significant testicular atrophy post cycle because he’s not using HCG (an injectable). Had you two agreed on HCG use, he probably would keep 4 to 8 pounds additional muscle, making his net gain anywhere from 14 to 20 pounds.
Underground steroid information from around the world
by Brock Strasser
Eating Your Liver With a Nice Chianti?
Q: My boyfriend has decided to try steroids for the first time after discussing them with me. While I’m grateful that our relationship is honest and open enough for him to talk to me about this, I’m concerned about his choice of steroids. We both decided that he shouldn’t use any injectable stuff, so please, let’s not even mention this.
My guy is planning on using 25 mg of D-bol for four weeks. Two weeks into this he plans to add 50 mg of something called Anapolan, which I heard was really liver toxic. He plans to stay on this for four weeks and two weeks into the Anapolan he’s planning to add in 30 mg of Winstrol taken orally for four weeks. Is this safe to his liver? I’ve read a lot of horrible things about oral steroid use being associated with liver cancer and cirrhosis. If this isn’t safe, what orals to you suggest instead of these?
A: For the longest time, I’ve been trying to dispel the urban myth that steroids cause liver cancer and liver damage. I’ve repeatedly written and stated that most people, especially if they don’t have any underlying health conditions, can use oral steroids for 10 to 12 weeks per year every year for a decade and not suffer from any lasting ill effects. Here’s a study I found that gives much credence to my "philosophy":
Anabolic steroid-induced hepatotoxicity: Is it overstated?
Clin J Sport Med 1999 Jan;9(1):34-9 (ISSN: 1050-642X)
Dickerman RD; Pertusi RM; Zachariah NY; Dufour DR; McConathy WJ
The Department of Biomedical Science, University of North Texas Health Science Center, Fort Worth 76107-2699, USA.
OBJECTIVE: There have been numerous reports of hepatic dysfunction secondary to anabolic steroid use based on elevated levels of serum aminotransferases. This study was conducted to distinguish between serum aminotransaminase elevations secondary to intense resistance training and anabolic steroid-induced hepatotoxicity in elite bodybuilders.
DESIGN: This was a case-control study of serum chemistry profiles from bodybuilders using and not using anabolic steroids with comparisons to a cohort of medical students and patients with hepatitis.
PARTICIPANTS: The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and nonexercising medical students (592) were used as controls.
MAIN OUTCOME MEASURES: The focus in blood chemistry profiles was aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels.
RESULTS: In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT.
CONCLUSION: Prior reports of anabolic steroid-induced hepatotoxicity based on elevated aminotransferase levels may have been overstated, because no exercising subjects, including steroid users, demonstrated hepatic dysfunction based on GGT levels. Such reports may have misled the medical community to emphasize steroid-induced hepatotoxicity when interpreting elevated aminotransferase levels and disregard muscle damage. For these reasons, when evaluating hepatic function in cases of anabolic steroid therapy or abuse, CK and GGT levels should be considered in addition to ALT and AST levels as essential elements of the assessment.
Now I’m sure that if someone "lived" on oral steroids (by oral, I’m referring to those that are alpha alkylated at carbon 17) week in and week out for years, he’d end up with serious health issues. But based on what you’ve written, I’d guess that your boyfriend (without knowing anything else about him) will handle this quite well and if he eats and trains correctly, he should gain upwards of 10 to12 pounds of solid muscle from this cycle.
I’d suggest he consider adding in an anti-aromatase (Arimidex) or anti-estrogen (Nolvadex, Clomid) during the time he’s on D-bol and for one week after.
He should also expect significant testicular atrophy post cycle because he’s not using HCG (an injectable). Had you two agreed on HCG use, he probably would keep 4 to 8 pounds additional muscle, making his net gain anywhere from 14 to 20 pounds.
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