Diet Critique

[mickyw2345]

Hi guys, just wanted to know if there was anything you tought i should change.

Stats:

21yo
16%BF
6'3"
220lbs

Meal 1 - 45g Oats

Meal 2 - 100g Tuna Steak, Salad

Meal 3 - SIS Rego Shake (314kcal, Carbohydrate 55g, Protein 27g, Fat trace).

Meal 4 - 60g Wholewheat Pasta or Basmati Rice
Tuna Steak/ Chicken Breast/ Steak

Meal 5 - Mixed Veggies
Tuna Steak/ Chicken Breast/ Steak

Meal 6 - Casein Shake


I usually alternate the sources of protein, weather it be steak, tuna or chicken, i used to drink alot of coffe/tea but i've swapped it for green tea apart from first thing in the mornig and one in the evening the coffee is decaff and i used a tsp of splenda.

I also take fish oil caps 3 times a day
and a standard multi vitamin

i seem to be loosing fat, but have noticed a drop in stregnth.

Any info's appreciated

Thanks.

 

[Suareezay]

is meal 1 a typo. wheres the protein?

dropping fat usually = strength loss, although you can continue adding weight to the bar for a while if you diet down slow, but eventually, youll start getting weaker.

 

[mickyw2345]

At the moment i just have oats for meal 1 but, i'm thinking of adding a protein shake with a tsp of udo's added in.

 

[Suareezay]

thatd probably be a good idea...why would you not have protein after not eating for 8+ hours?

 

[macro]

green tea has a lot heath benefits, in particular aiding metabolically like this study points out

Obes Res. 2005 Jun;13(6):982-90. Related Articles, Links


Green tea polyphenol epigallocatechin gallate inhibits adipogenesis and induces apoptosis in 3T3-L1 adipocytes.

Lin J, Della-Fera MA, Baile CA.

444 Edgar L. Rhodes Center for Animal and Dairy Science, University of Georgia, Athens, GA 30602-2771, USA.

OBJECTIVE: Green tea catechins have been shown to promote loss of body fat and to inhibit growth of many cancer cell types by inducing apoptosis. The objective of this study was to determine whether epigallocatechin gallate (EGCG), the primary green tea catechin, could act directly on adipocytes to inhibit adipogenesis and induce apoptosis. RESEARCH METHODS AND PROCEDURES: Mouse 3T3-L1 preadipocytes and mature adipocytes were used. To test the effect of EGCG on viability, cells were incubated for 3, 6, 12, or 24 hours with 0, 50, 100, or 200 microM EGCG. Viability was quantitated by MTS assay. To determine the effect of EGCG on apoptosis, adipocytes were incubated for 24 hours with 0 to 200 microM EGCG, then stained with annexin V and propidium iodide and analyzed by laser scanning cytometry. Both preadipocytes and adipocytes were also analyzed for apoptosis by terminal deoxynucleotidyl transferase dUTP nick-end labeling assay. To determine the effect of EGCG on adipogenesis, maturing preadipocytes were incubated during the 6-day induction period with 0 to 200 microM EGCG, then stained with Oil-Red-O and analyzed for lipid content. RESULTS: EGCG had no effect on either viability or apoptosis of preconfluent preadipocytes. EGCG also did not affect viability of mature adipocytes; however, EGCG increased apoptosis in mature adipocytes, as demonstrated by both laser scanning cytometry and terminal deoxynucleotidyl transferase dUTP nick-end labeling assays. Furthermore, EGCG dose-dependently inhibited lipid accumulation in maturing preadipocytes. DISCUSSION: These results demonstrate that EGCG can act directly to inhibit differentiation of preadipocytes and to induce apoptosis of mature adipocytes and, thus, could be an important adjunct in the treatment of obesity.


best price on high EGCG, Green tea extract
https://www.anabolicfitness.net/shop/product_info.php?id=121