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This only applies to women.
Women's bodies convert most of the testosterone produced into estrogen. So any access taken such as a steroid, will be converted into estrogen. That is why most women will experience bloating and fat gain when taking a steroid.
The androgens are converted into estrogen. Essentially what women do when they take a steroid, is pump their bodies full of estrogen. So, to get what you want from a cycle is to use an Aromatase inhibitor (AI) along with the steroid. What would be the point of taking an anabolic if most of it will be converted into estrogen anyway? Making you gain fat and a ton of water?
In regards to taking Nolvadex or Arimidex or Femara to combat conversion of androgens into estrogen, it would be, in my opinion, best to take Arimidex or Femara in low doses. Femara is a very powerful anti estrogen and the half life is about 40hours, so those daily doses will build up. You can take .25mg of Femara per day along with whatever steroid compound to reduce and eliminate the conversion of androgens in your blood into estrogen, do not use Nolvadex. (Half life of Femara is 2days, that means half the hormone is active in the body after two days). Play with the dosages, may try to start with 1.25 (skipping every two days). If you read in the articles forum a thread I started called "NOLVADEX", Nolvadex competes with the estrogen receptor, it does not stop the conversion of androgens into estrogen. So when you stop taking NOLVADEX, you still have a flood of estrogen in your system that will flood your reseptors when Nolvadex is discontinued. The conversion of androgens (steroids) into estrogen is what causes the fat gain and water gain in women. Arimidex in this case stops that conversion of androgens into estrogen, therefor no bloating or fat gain will occur. The dose of the steroid taken may also be reduced if taken with an AI; reduce the dose only as needed (to avoid any out-coming side effects).
How an Aromatase inhibitor (AI) works.
An Aromatase inhibitor (AI) binds to the aromatase enzyme and blocks it from converting androgens to estrogen.
In women, estrogens are mainly derived from the action of the aromatase enzyme, which converts adrenal androgens (primarily androstenedione and testosterone) to estrone and estradiol. Letrozole is a nonsteroidal competitive inhibitor of the aromatase enzyme system; it inhibits the conversion of androgens to estrogens. Letrozole is as effective in reducing uterine weight, elevating serum LH. Letrozole does not lead to an increase in serum FSH. Letrozole selectively inhibits gonadal steroidogenesis but has no significant effect on adrenal mineralocorticoid or glucocorticoid synthesis. Letrozole inhibits the aromatase enzyme by competitively binding to the heme of the cytochrome P450 subunit of the enzyme, resulting in a reduction of estrogen biosynthesis in all tissues. Treatment of women with letrozole significantly lowers serum estrone, estradiol and estrone sulfate and has not been shown to significantly affect adrenal corticosteroid synthesis, aldosterone synthesis, or synthesis of thyroid hormones.
Women's bodies convert most of the testosterone produced into estrogen. So any access taken such as a steroid, will be converted into estrogen. That is why most women will experience bloating and fat gain when taking a steroid.
The androgens are converted into estrogen. Essentially what women do when they take a steroid, is pump their bodies full of estrogen. So, to get what you want from a cycle is to use an Aromatase inhibitor (AI) along with the steroid. What would be the point of taking an anabolic if most of it will be converted into estrogen anyway? Making you gain fat and a ton of water?
In regards to taking Nolvadex or Arimidex or Femara to combat conversion of androgens into estrogen, it would be, in my opinion, best to take Arimidex or Femara in low doses. Femara is a very powerful anti estrogen and the half life is about 40hours, so those daily doses will build up. You can take .25mg of Femara per day along with whatever steroid compound to reduce and eliminate the conversion of androgens in your blood into estrogen, do not use Nolvadex. (Half life of Femara is 2days, that means half the hormone is active in the body after two days). Play with the dosages, may try to start with 1.25 (skipping every two days). If you read in the articles forum a thread I started called "NOLVADEX", Nolvadex competes with the estrogen receptor, it does not stop the conversion of androgens into estrogen. So when you stop taking NOLVADEX, you still have a flood of estrogen in your system that will flood your reseptors when Nolvadex is discontinued. The conversion of androgens (steroids) into estrogen is what causes the fat gain and water gain in women. Arimidex in this case stops that conversion of androgens into estrogen, therefor no bloating or fat gain will occur. The dose of the steroid taken may also be reduced if taken with an AI; reduce the dose only as needed (to avoid any out-coming side effects).
How an Aromatase inhibitor (AI) works.
An Aromatase inhibitor (AI) binds to the aromatase enzyme and blocks it from converting androgens to estrogen.
In women, estrogens are mainly derived from the action of the aromatase enzyme, which converts adrenal androgens (primarily androstenedione and testosterone) to estrone and estradiol. Letrozole is a nonsteroidal competitive inhibitor of the aromatase enzyme system; it inhibits the conversion of androgens to estrogens. Letrozole is as effective in reducing uterine weight, elevating serum LH. Letrozole does not lead to an increase in serum FSH. Letrozole selectively inhibits gonadal steroidogenesis but has no significant effect on adrenal mineralocorticoid or glucocorticoid synthesis. Letrozole inhibits the aromatase enzyme by competitively binding to the heme of the cytochrome P450 subunit of the enzyme, resulting in a reduction of estrogen biosynthesis in all tissues. Treatment of women with letrozole significantly lowers serum estrone, estradiol and estrone sulfate and has not been shown to significantly affect adrenal corticosteroid synthesis, aldosterone synthesis, or synthesis of thyroid hormones.
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