do what you see is doing good for you and suites your situation ,body and needs. dont experiment un-necessarily. i know you are running from test but if you feel like shit because lack of androgen anywhere between the cycle as you dont have test add some proviron.. 25 -50 mg/day would be enough giving extra strength..and intensity.
this statement is not supported by any medical data I can find. or by my experience. although it may make him think he is ok even though his T levels are in the toilet, you are wrong about the strenght and intensity. this is noted in my following post, quotation, an explanation on some studies on proviron.
test is very very natural for the body..it does all the function which other dug just cant.but you can live without it for 6-7 if you are smart enough . and you seems smart
...you are gona have some 90% of your strenght gain from anavar post cycle...and also the lean muscle gain( if you eat shit of food)...it aint going no where
..
this is your opinion and is worthless
ps:if i was in your situation i would have run dbol with proviron to keep the strenght high and water bloat down.
again you are making claims that proviron actually has some effect on aromtizing compounds, the following studies show otherwise. If it did, the TT levels would have changed due to inhibiton of aromatase, in the androgen therapy studies.
no need to run any oral over long period of time..after few weeks there effectiveness diminish . short burst cycle is fine. you wont feel depression after this short busrt cycle with weak compound..and guess what you are doing great and thats count
now your implying that he will have no suppression, which you have not supported yet. maybe you supported the fact that HPTA will not be suppressed by proviron, but im still not sold. you did say that 85 men experienced HPTA suppression from proviron right? not to mention that scally doesnt buy the study either. not to mention we CAN prove both dbol and Anavar (var) will suppress, so again worthless.
proviron = increased strength =increase poundage=increase lean muscle mass (if eat right..if then its very little) ..but he is not looking for mass...and proviron provides such pure strength and little polishing effect on the body that user think from where the hell i am seeing these strength gains as i am seeing no muscle or mass gain at all.
anavar= strength increase with adipose fat loss= more room for lean muscle at the same body weight =much stronger then the same weight category dude...
and thats his mission.
proviron for his brain to think clear and positive and give him little strength with vigor he might lack when/if test is suppressed and not supplied from outside.. . anavar make some ppl lethargic when taken alone..proviron will tackle this psychological/physiological problem if he experience it which i highly doubt considering his young age.
your strength claims are not supported are not supported by the following argument, which is backed up scientifically.
from Dr. Scally
This is a post on placed on another thread concerning the same topic.
Dianabol (methandione, methandrostenolone, metandienone, and a host of other names) suppresses the HPTA. The use of dianabol in the hope that it will provide HPTA normalization is misguided. More details, later, can be provided, if requested.
However, a brief note on proviron. What evidence is there that proviron lacks androgenic activity. The literature presents this by the absence of proviron to influence significantly infertility, erythropoiesis, lipids, and sex hormones. Except for the obsessive compulsive that needs to take a substance, thus replacing an AAS with adverse HPTA effects with one that does not, proviron is a worthless AAS, useful for nothing. Proviron will not support or provide any basis for the return of HPTA function.
The quoted abstract from the study by Varma and Patel really does not give one any information. [Varma TR, Patel RH. The effect of mesterolone on sperm count, on serum follicle stimulating hormone, luteinizing hormone, plasma testosterone and outcome in idiopathic oligospermic men. Int J Gynaecol Obstet 1988;26:121-8.]
The study is poor from the abstract alone. Please note that the statement, "Mesterolone had no depressing effect on low or normal serum FSH and LH levels but had depressing effect on 25% if the levels were elevated," refers unidentified group. The groups in the study include, "One hundred ten patients . . . had normal serum FSH, LH and plasma testosterone, 85 patients . . . had low serum FSH, LH and low plasma testosterone." Nowhere is there a group with elevated levels. Nonetheless, the cited effect is a "depressing effect" not stated as significant. Knowing the fluctuation in gonadotropin levels on testing even at a P<0.05 would not be meaningful. But it does go to the point that proviron has no adverse effect on the HPTA.
Mesterolone is useless for infertility. A year after the Varma study, 1989, the World Health Organization published a study demonstrating, "[n]o significant changes semen quality during the course of the study, apart from an increase in sperm concentration 3 months after the start of treatment. The increase was greatest among the placebo treated group, but did not differ significantly between treatment groups." [Mesterolone and idiopathic male infertility: a double-blind study. World Health Organization Task Force on the Diagnosis and Treatment of Infertility. Int J Androl 1989;12:254-64.]
In 1991, a study concludes, "Because similar semen improvement also occurred in the placebo controls, our findings cast doubt on the possible usefulness of high-dose Mesterolone treatment of idiopathic male infertility." [Gerris J, Comhaire F, Hellemans P, Peeters K, Schoonjans F. Placebo-controlled trial of high-dose Mesterolone treatment of idiopathic male infertility. Fertil Steril 1991;55:603-7.]
These confirm an earlier study from 1983. [Wang C, Chan CW, Wong KK, Yeung KK. Comparison of the effectiveness of placebo, clomiphene citrate, mesterolone, pentoxifylline, and testosterone rebound therapy for the treatment of idiopathic oligospermia. Fertil Steril 1983;40:358-65.] Treatment with the mesterolone (100 mg/day) therapy did not result in a significant increase in the mean sperm concentration or pregnancy in the partners.
Proviron is useless in promoting erythropoiesis (formation of red blood cell elements) and bone formation (a mixed effect of testosterone through the androgen receptor and estradiol receptor), both evidence of androgenic activity. Mesterolone (100 mg/d) is ineffective in raising hemoglobin and hematocrit levels significantly from baseline in individuals with hypogonadism.
The study cites that Mesterolone did not increase serum testosterone (but also did not mention that there is a decrease). [Jockenhovel F, Vogel E, Reinhardt W, Reinwein D. Effects of various modes of androgen substitution therapy on erythropoiesis. Eur J Med Res 1997;2:293-8.]
As recent as 2003, mesterolone (100 mg/d) for 6 months administered to hypogonadal males failed to significantly raise bone mineral density (BMD). Treatment with testosterone undecanoate (160 mg/d), testosterone enanthate 250 mg (every 21 days), or a single subcutaneous implantation of 1,200 mg crystalline testosterone did result in BMD increases. [Schubert M, Bullmann C, Minnemann T, Reiners C, Krone W, Jockenhovel F. Osteoporosis in male hypogonadism: responses to androgen substitution differ among men with primary and secondary hypogonadism. Horm Res 2003;60:21-8.]
Erythropoiesis and bone formation are positive aspects of androgens useful under certain clinical conditions. AAS consistently have adverse effects on lipid profiles that are generally observed as a decrease in HDL (good cholesterol). In 1999, twenty years after the study cited by MaxRep [Nikkanen V. Plasma cholesterol, triglycerides, FSH and testosterone levels of normolipemic male patients with decreased fertility treated with mesterolone. Andrologia 1979;11:33-6.] proviron was found to adversely effect the lipid profile in hypogonadal men. The study by abstract analysis is hard to detail but an adverse effect of proviron is reported. Also, the study reports on serum testosterone levels with androgen treatments. Androgen substitution led to no significant increase of serum testosterone in the proviron group, subnormal testosterone in the testosterone undecanoate group, normal testosterone in the testosterone enanthate group, and high-normal testosterone in the crystalline testosterone group. The message is proviron did not affect the HPTA. [Jockenhovel F, Bullmann C, Schubert M, et al. Influence of various modes of androgen substitution on serum lipids and lipoproteins in hypogonadal men. Metabolism 1999;48:590-6.] The same author reports that proviron administration has no effect on serum FSH or testosterone. [Nikkanen V. The effects of mesterolone on the male accessory sex organs, on spermiogram, plasma testosterone and FSH. Andrologia 1978;10:299-306.]
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I have said too much already. A further review of proviron literature will not change the use of proviron as an AAS for either anabolic or androgenic effects. Bottom line: Proviron is of no use for anything.
Thank you."