This may sound like an immature or stupid question, but can taking AAS during your teens change your facial structure? Such as masculine features like a larger jaw. And if it does, is it enough to be noticeable? I know hgh can change your facial structure, jay curlers head looks like a jacked banana.
Can AAS Change Facial Structure
- Thread starter Sikkent
- Start date
ksvc
New member
Androgens are part of a multifaceted anabolic mechanism that occurs during puberty. They play a role in both growth and virilization.
Sex steroids, growth hormone, insulin-like growth factor-1: neuroendocrine and metabolic regulation in puberty.
Mauras N1, Rogol AD, Haymond MW, Veldhuis JD.
Author information
Abstract
The control of the onset of puberty involves the complex interaction of pituitary and gonadal hormones. At a preprogrammed time in a child's life there is an increase in the amplitude of GnRH pulses which triggers a cascade of events including increases in the amplitude of FSH and LH pulses, followed by marked increases in gonadal sex steroidal output, which in turn increases growth hormone (GH) and insulin-like growth factor-1 (IGF-1) production. Evidence suggests that there is an integral interaction between the endogenous opiate system and the hypothalamic-pituitary-gonadal axis, at least in the later stages of puberty in the male. Both androgenic and estrogenic hormones markedly increase GH production rates as measured by deconvolution models in the prepubertal human, and compelling data strongly suggest that it is indeed the estrogen which controls the feedback amplification of GH production during puberty even in the male. It appears that the prepubertal gonad is actively producing sex hormones which might be important in the control of GH production since early childhood. The translation of these neuroendocrine rhythms into distal metabolic actions is also reviewed. Utilizing isotopic tracer infusions of the essential amino acid leucine, studies clearly show a selective stimulation of whole body protein synthesis by both GH and IGF-1. GH, IGF-1 and androgenic hormones all increase in puberty, stimulating whole body protein anabolism during that period. However, we observed no protein-anabolic effect in the hypogonadal female given increasing doses of estrogen. The latter suggests that at least as it pertains to whole body protein effects, the action of androgens is probably mediated via the androgen and not the estrogen receptor, in clear distinction from the estrogen-mediated effects of androgens on the neuroendocrine axis. Calcium absorption and retention are also positively affected by the androgens as shown by significant increases in calcium absorption and retention after the administration of testosterone to the prepubertal male. This suggests an important role of sex steroidal hormones in the mineralization of the skeleton. In conclusion: GH, IGF-1 and sex steroids all markedly increase during puberty and their actions are amplified mutually as they control growth, increase muscle mass and affect the mineralization of the skeleton. The dichotomy of androgen and estrogen effects in the male and female may regulate the differential timing of the onset of puberty and final height in the two sexes. The synergistic actions of these anabolic hormones appear to be most significant during the finite years of puberty.
Sex steroids, growth hormone, insulin-like growth factor-1: neuroendocrine and metabolic regulation in puberty.
Mauras N1, Rogol AD, Haymond MW, Veldhuis JD.
Author information
Abstract
The control of the onset of puberty involves the complex interaction of pituitary and gonadal hormones. At a preprogrammed time in a child's life there is an increase in the amplitude of GnRH pulses which triggers a cascade of events including increases in the amplitude of FSH and LH pulses, followed by marked increases in gonadal sex steroidal output, which in turn increases growth hormone (GH) and insulin-like growth factor-1 (IGF-1) production. Evidence suggests that there is an integral interaction between the endogenous opiate system and the hypothalamic-pituitary-gonadal axis, at least in the later stages of puberty in the male. Both androgenic and estrogenic hormones markedly increase GH production rates as measured by deconvolution models in the prepubertal human, and compelling data strongly suggest that it is indeed the estrogen which controls the feedback amplification of GH production during puberty even in the male. It appears that the prepubertal gonad is actively producing sex hormones which might be important in the control of GH production since early childhood. The translation of these neuroendocrine rhythms into distal metabolic actions is also reviewed. Utilizing isotopic tracer infusions of the essential amino acid leucine, studies clearly show a selective stimulation of whole body protein synthesis by both GH and IGF-1. GH, IGF-1 and androgenic hormones all increase in puberty, stimulating whole body protein anabolism during that period. However, we observed no protein-anabolic effect in the hypogonadal female given increasing doses of estrogen. The latter suggests that at least as it pertains to whole body protein effects, the action of androgens is probably mediated via the androgen and not the estrogen receptor, in clear distinction from the estrogen-mediated effects of androgens on the neuroendocrine axis. Calcium absorption and retention are also positively affected by the androgens as shown by significant increases in calcium absorption and retention after the administration of testosterone to the prepubertal male. This suggests an important role of sex steroidal hormones in the mineralization of the skeleton. In conclusion: GH, IGF-1 and sex steroids all markedly increase during puberty and their actions are amplified mutually as they control growth, increase muscle mass and affect the mineralization of the skeleton. The dichotomy of androgen and estrogen effects in the male and female may regulate the differential timing of the onset of puberty and final height in the two sexes. The synergistic actions of these anabolic hormones appear to be most significant during the finite years of puberty.
Megatron28
Moderator
Have you completed puberty yet? How old are you?
Megatron28
Moderator
You should wait until you are 25. Let your brain/endocrine system finish developing.
Chicago712
Pro Bodybuilder
Listen to this, I started using at 19 and I'm 25 now and I'm in the process of getting on trt for the rest of my life..
On top of that I've lost one of my true loves this last past year due to my steroid use and how some compounds effect me
Megatron28
Moderator
You always take a risk when you cycle. Make sure you fully understand all the risks before you make a decision. You can't unring the bell as they say.
Chicago712
Pro Bodybuilder
Nope never messed anything up, I even took a more aggressive approach , I had blood work, complete pcts, and way more time off then on
And I sit here today with a 315 test level
PrinceDianabol
Well-known member
Yeah bro there is ALWAYS a risk when you cycle, doesn't matter how good you do your PCT and how light or small the cycles are. Once you shutdown, the risk is there. In males under 25 (approximately that age anyway) the risk is much much greater. I also started at age 19, since then I've had a whole host of problems arise... Not to get too far into it, but I developed performance anxeity (dick doesn't work - but its all in my head) aswell as a few other problems. The brain is an amazing thing man, and definetly should not be fucked with while its still devloping. Remember when you cycle your fuckin with your hormones - they play a massive role in your devlopment.
Do what you wanna do, but take it from us... If we had a second chance, we'd go back and wait til we were older... Any day.
Do what you wanna do, but take it from us... If we had a second chance, we'd go back and wait til we were older... Any day.
PrinceDianabol
Well-known member
How do you know they're fine? Do you think they'd really tell you if there dick stopped working? Do they have bloodwork to show they're fine? Even if they do, its not uncommon for problems to arise further down the track. In a year or two times they might 'randomly' become hypogonadal.
Its because until the age of 25, your HPTA system is still devloping and 'learning' what its doing. When you shut it down and send your hormones way out of whack, it gets confused as to how muhh its supposed to produce etc. very little to do with genetics, everyone has to develop their HPTA.
PrinceDianabol
Well-known member
Double post
I just watched a vid on Dorian Yates and he said he was on steroids for 12 years and his doctors said he's healthy for his age. Shouldn't him and all the other old school pros be dead? They were on way more than I could ever imagine taking, id do at most two light cycles of just test.
Megatron28
Moderator
My 80 ywar old uncle smoked two packs of cigs every day since he was 14. Drank every day too. No cancer. Heart still going. Liver is great. I guess anyone can do all that if he is fine. Right?
Also, there is a big difference between dead and messed up. Can you name the risks of using AAS?
PrinceDianabol
Well-known member
More than likely they are all on TRT though. Which isn't 'unhealthy,' but being hypogonadal is. Meaning if they don't keep injecting a cruise dose for the rest of their lives up til the day they die, they will be unhealthy/unfit/depressed/lethargic etc. etc.
