halfwit
I like turtles.
I want to state that I recall seeing similar flaws in many studies where the researchers indeed provide poor control. The fault actually lies with the peer groups that enables such studies to be allowed without first providing a disclaimer of such. This extends into so many areas of science, it's not even funny.
With that said, I do want to add that for catabolism to occur, the body must be out of sufficient energy AND building blocks (macronutrients) for repair during sleep. As the liver stores glycogen before we go into a fasted state, we have energy reserves covered (this is a VERY important concept as it's why we don't die from hypoglycemia in our sleep) on top of the ability to tap into body fat stores. For de novo gluconeogenesis to occur, there must be a depletion of both glycogen and reserves in order to destroy muscle for energy. (think keto dieting with insufficient fat intake)
Please don't make my tired butt cite a reference.
I enjoy a healthy debate, and I ALWAYS love learning. In this case, unfortunately we have to go by anecdotal evidence as I too have yet to see a direct casual relationship between AA absorption and actual hypertrophic response/recovery. I can state from experience however, that the slower digesting proteins do provide a better measure of satiety, which helps keep those hunger pangs at bay, especially if we awaken in the middle of the night. I like to think it provides a measure of "protection" for my hard-earned muscle mass, but I can't prove it.
My .02 modest (I'm a hobbyist that loves the lifestyle, my research skills come purely from an entirely different field ) cents.
With that said, I do want to add that for catabolism to occur, the body must be out of sufficient energy AND building blocks (macronutrients) for repair during sleep. As the liver stores glycogen before we go into a fasted state, we have energy reserves covered (this is a VERY important concept as it's why we don't die from hypoglycemia in our sleep) on top of the ability to tap into body fat stores. For de novo gluconeogenesis to occur, there must be a depletion of both glycogen and reserves in order to destroy muscle for energy. (think keto dieting with insufficient fat intake)
Please don't make my tired butt cite a reference.
I enjoy a healthy debate, and I ALWAYS love learning. In this case, unfortunately we have to go by anecdotal evidence as I too have yet to see a direct casual relationship between AA absorption and actual hypertrophic response/recovery. I can state from experience however, that the slower digesting proteins do provide a better measure of satiety, which helps keep those hunger pangs at bay, especially if we awaken in the middle of the night. I like to think it provides a measure of "protection" for my hard-earned muscle mass, but I can't prove it.
My .02 modest (I'm a hobbyist that loves the lifestyle, my research skills come purely from an entirely different field ) cents.