testosterone level is what was down on my sheet, i know that a few levels come back becasue when i went a few months back he had a few readings on the screen, not just the 12.8 or in this case a 36.0
the problem here is as of 6pm today (2 and a half hours away here in the uk) it will have been exactly 8 days since my last 2000iu shot, really unhappy with my doctors for not giving me the urget call friday.
i dont want to waste the clomid by taking it now if the hcg has worn off slightly.
the test result is obviously showing that as of tuesday 15th march around 11am my testosterone level was 36.0. How long is it in the system for?
Id rather wait and run HCG again if possible, if it means a better chance of success.
you need the hcg out of your system to start clomid/nolva
you might have made the situation owrse by not having an Aromatase inhibitor (AI) with the HCG, thus increasing your estrogen levels which negatively effect your hpta feedback mechanism
its time to start the serms
heres a good study of long term users getting back to normal
STREET C, SCALLY MC. Pharmaceutical Intervention of Anabolic Steroid Induced
Hypogonadism - Our Success at Restoration of the HPG Axis. Medicine and Science in Sports
and Exercise 2000;32(5)Suppl.
High-dose anabolic androgenic steroid (AAS) administration results in hypogonadotropic
hypogonadism (HH). Physical manifestations can include one or more of the following:
depression, decreased sexual desire, impotence, feelings of apathy, testicular atrophy, and loss of
muscle mass and strength. Due to feedback inhibition, laboratory values drop well below
established physiologic norms: luteinizing hormone (LH) >3.6 IU/L, follicle stimulating
hormone (FSH) >2.25 IU/L, and testosterone (T) >300 ng/dL. A search of the literature reveals
an absence of studies dealing specifically with AAS induced HH, and restoration of normal
endocrine function. We report on two interesting cases of AAS using bodybuilders who were
brought out of the hypogonadal state. Blood samples were taken in the morning for both subjects
and analyzed using chemiluminescence (Quest Diagnostics, Irvine, TX). Post-therapy samples
were taken 15 days after the last hCG injection.
Case 1: 6'0" 206 lbs. 33 yr old Caucasian male
with a 10+ year history of steroid self-administration for bodybuilding and powerlifting. By his
own admission he was a "heavy" user, taking from 500 mg/wk to 2+ grams/wk. Pre-treatment
values: LH < 1.0 IU/L, T 191 ng/dL. One course of therapy (32 days) was given: 2,500 IU of
hCG every 4 days (8 injections total), 50 mg clomiphene bid and 10 mg tamoxifen qd. Despite
massive drug use patient was an exceptionally good responder. Post-treatment values: LH 5.2
IU/L, T 1072 ng/dL.
Case 2: 5'10" 184 lbs 36 yr old Caucasian male with a 2 yr history of
continuous nandrolone use (200-400 mg/wk). Pre-values: LH < 1.0 IU/L, T 45 ng/dL.
Treat 1
(32 days): 2,500 IU hCG every 4 d (8 total), clomiphene (50 mg bid) and arimidex (1 mg qd).
Post-values: LH < 1.0 IU/L, T 38 ng/dL.
Treat 2 (60 days): 5,000 IU hCG every 4 days (4 inj
total) followed by 2,500 IU hCG every 4 d (4 inj total), clomiphene (50 mg bid) and tamoxifen
(10 mg qd). Post-values: LH > 1.4 IU/L, T 63 ng/dL.
Treat 3 (32 days): 5,000 IU hCG qod (6 inj
total) followed by 2,500 IU hCG qod (6 inj total) given simultaneously with menotropins 150 IU
qod (6 inj total), clomiphene (50 mg bid) and tamoxifen (10 mg bid). Post-values: LH 9.8 IU/L,
T 507 ng/dL.
Restoration of the HPG axis, even in severe cases of hypogonadism, is possible
with combined therapies and careful monitoring of the patient. With continued popularity of
these drugs, long-term androgen deficiency is a health concern for former AAS users. Further
research is needed in this area.
