Doesnt work that way. At the completion of a cycle, androgen levels are falling rapidly. The body compensates with an over production of estrogen in order to upregulate the androgen receptor (make them more sensitive to androgens). Like androgens, estrogen negatively impacts the HTPA. By blocking this estrogen with SERMs, both androgen and estrogen levels will be low. Low levels of each stimulate the hyothalamus to produce GnRH. The pituitary responds to the GnRH by producing LH. LH stimulates the leydig cells to produce testosterone.