swellonemm
New member
neg rep, surf dog is all u can come back with lil man. thanks!
testosterone/winstrol cycle
- Thread starter ricbjj
- Start date
swellonemm
New member
real mature....lol
DADAWG
Community Veteran
i did that not him , keep being a asshole and it will get worse.
swellonemm
New member
lol, id rather give the correct info to this guy for best results instead of being nice. Relax big dawg, id just tryin to help these first timers. i think people get influenced to easily and end up doing the wrong thing sometimes ya know. sometime u have to talk to em like this. sorry though didnt know it was that serious.
THE-DET-OAK
IncreasedMyT @ ULV
why do they make 24 hour claritin D and 12 hour?
THE-DET-OAK
IncreasedMyT @ ULV
just so you know your theory is jacked-and the idea that the reason they made different esters is becuase of there different effects on muscle growth and water retention couldnt be further from the truth. It was a simple matter of experimentation.
here is a lil history lesson mr. know it all.
Wikepedia
History
A testicular action was linked to circulating blood fractions – now understood to be a family of androgenic hormones – in the early work on castration and testicular transplantation in fowl by Arnold Adolph Berthold (1803–1861).[68] Research on the action of testosterone received a brief boost in 1889, when the Harvard professor Charles-Édouard Brown-Séquard (1817–1894), then in Paris, self-injected subcutaneously a “rejuvenating elixir” consisting of an extract of dog and guinea pig testicle. He reported in The Lancet that his vigor and feeling of well-being were markedly restored but, predictably, the effects were transient[69] (and likely based on a placebo effect), and Brown-Séquard’s hopes for the compound were dashed. Suffering the ridicule of his colleagues, his work on the mechanisms and effects of androgens in human beings was abandoned by Brown-Séquard and succeeding generations of biochemists for nearly 40 years.
The trail remained cold until the University of Chicago’s Professor of Physiologic Chemistry, Fred C. Koch, established easy access to a large source of bovine testicles—the Chicago stockyards—and to students willing to endure the ceaseless toil of extracting their isolates. In 1927, Koch and his student, Lemuel McGee, derived 20 mg of a substance from a supply of 40 pounds of bovine testicles that, when administered to castrated roosters, pigs and rats, remasculinized them.[70] The group of Ernst Laqueur at the University of Amsterdam purified testosterone from bovine testicles in a similar manner in 1934, but isolation of the hormone from animal tissues in amounts permitting serious study in humans was not feasible until three European pharmaceutical giants—Schering (Berlin, Germany), Organon (Oss, Netherlands) and Ciba (Basel, Switzerland)—began full-scale steroid research and development programs in the 1930’s.
The Organon group in the Netherlands were the first to isolate the hormone, identified in a May 1935 paper "On Crystalline Male Hormone from Testicles (Testosterone)".[71] They named the hormone testosterone, from the stems of testicle and sterol, and the suffix of ketone. The structure was worked out by Schering’s Adolf Butenandt.[72]
The chemical synthesis of testosterone was achieved in August that year, when Butenandt and G. Hanisch published a paper describing "A Method for Preparing Testosterone from Cholesterol." Only a week later, the Ciba group in Zurich, Leopold Ruzicka (1887–1976) and A. Wettstein, announced a patent application in a paper "On the Artificial Preparation of the Testicular Hormone Testosterone (Androsten-3-one-17-ol)." These independent partial syntheses of testosterone from a cholesterol base earned both Butenandt and Ruzicka the joint 1939 Nobel Prize in Chemistry.[73][74] Testosterone was identified as 17β-hydroxyandrost-4-en-3-one (C19H28O2), a solid polycyclic alcohol with a hydroxyl group at the 17th carbon atom. This also made it obvious that additional modifications on the synthesized testosterone could be made, i.e., esterification and alkylation.
The partial synthesis in the 1930s of abundant, potent testosterone esters permitted the characterization of the hormone’s effects, so that Kochakian and Murlin (1936) were able to show that testosterone raised nitrogen retention (a mechanism central to anabolism) in the dog, after which Allan Kenyon’s group[75] was able to demonstrate both anabolic and androgenic effects of testosterone propionate in eunuchoidal men, boys, and women. The period of the early 1930s to the 1950s has been called "The Golden Age of Steroid Chemistry",[76] and work during this period progressed quickly. Research in this golden age proved that this newly synthesized compound—testosterone—or rather family of compounds (for many derivatives were developed from 1940 to 1960), was a potent multiplier of muscle, strength, and wellbeing.[58]
here is a lil history lesson mr. know it all.
Wikepedia
History
A testicular action was linked to circulating blood fractions – now understood to be a family of androgenic hormones – in the early work on castration and testicular transplantation in fowl by Arnold Adolph Berthold (1803–1861).[68] Research on the action of testosterone received a brief boost in 1889, when the Harvard professor Charles-Édouard Brown-Séquard (1817–1894), then in Paris, self-injected subcutaneously a “rejuvenating elixir” consisting of an extract of dog and guinea pig testicle. He reported in The Lancet that his vigor and feeling of well-being were markedly restored but, predictably, the effects were transient[69] (and likely based on a placebo effect), and Brown-Séquard’s hopes for the compound were dashed. Suffering the ridicule of his colleagues, his work on the mechanisms and effects of androgens in human beings was abandoned by Brown-Séquard and succeeding generations of biochemists for nearly 40 years.
The trail remained cold until the University of Chicago’s Professor of Physiologic Chemistry, Fred C. Koch, established easy access to a large source of bovine testicles—the Chicago stockyards—and to students willing to endure the ceaseless toil of extracting their isolates. In 1927, Koch and his student, Lemuel McGee, derived 20 mg of a substance from a supply of 40 pounds of bovine testicles that, when administered to castrated roosters, pigs and rats, remasculinized them.[70] The group of Ernst Laqueur at the University of Amsterdam purified testosterone from bovine testicles in a similar manner in 1934, but isolation of the hormone from animal tissues in amounts permitting serious study in humans was not feasible until three European pharmaceutical giants—Schering (Berlin, Germany), Organon (Oss, Netherlands) and Ciba (Basel, Switzerland)—began full-scale steroid research and development programs in the 1930’s.
The Organon group in the Netherlands were the first to isolate the hormone, identified in a May 1935 paper "On Crystalline Male Hormone from Testicles (Testosterone)".[71] They named the hormone testosterone, from the stems of testicle and sterol, and the suffix of ketone. The structure was worked out by Schering’s Adolf Butenandt.[72]
The chemical synthesis of testosterone was achieved in August that year, when Butenandt and G. Hanisch published a paper describing "A Method for Preparing Testosterone from Cholesterol." Only a week later, the Ciba group in Zurich, Leopold Ruzicka (1887–1976) and A. Wettstein, announced a patent application in a paper "On the Artificial Preparation of the Testicular Hormone Testosterone (Androsten-3-one-17-ol)." These independent partial syntheses of testosterone from a cholesterol base earned both Butenandt and Ruzicka the joint 1939 Nobel Prize in Chemistry.[73][74] Testosterone was identified as 17β-hydroxyandrost-4-en-3-one (C19H28O2), a solid polycyclic alcohol with a hydroxyl group at the 17th carbon atom. This also made it obvious that additional modifications on the synthesized testosterone could be made, i.e., esterification and alkylation.
The partial synthesis in the 1930s of abundant, potent testosterone esters permitted the characterization of the hormone’s effects, so that Kochakian and Murlin (1936) were able to show that testosterone raised nitrogen retention (a mechanism central to anabolism) in the dog, after which Allan Kenyon’s group[75] was able to demonstrate both anabolic and androgenic effects of testosterone propionate in eunuchoidal men, boys, and women. The period of the early 1930s to the 1950s has been called "The Golden Age of Steroid Chemistry",[76] and work during this period progressed quickly. Research in this golden age proved that this newly synthesized compound—testosterone—or rather family of compounds (for many derivatives were developed from 1940 to 1960), was a potent multiplier of muscle, strength, and wellbeing.[58]
swellonemm
New member
from ALL my experience, prop is a better testosterone for lean gains, deff better blood leve stability, and MOST deff. less water retention. this is why i use prop for npp cycles and say cyp for deca cycles and maybe enath for tren e. Know what i mean just logical for better gains. claritin thats a good one...lol seriously it made me laugh nice analogy.
THE-DET-OAK
IncreasedMyT @ ULV
i will agree-unlike most-that prop is more of a lean test-just had to point out your theory was a lil.........lacking.
I think its becuase of unstable blood levels, thats my theory anyway, and that its easier to attain stable levels with shorter esters. although most would say its easier to sustain stable blood levels with long esters, from my research i would theorize the opposite. it is just a theory though.
I think its becuase of unstable blood levels, thats my theory anyway, and that its easier to attain stable levels with shorter esters. although most would say its easier to sustain stable blood levels with long esters, from my research i would theorize the opposite. it is just a theory though.
THE-DET-OAK
IncreasedMyT @ ULV
DADAWG brought up some other factors that also play a role in it the other day, most people have no problem running a gram a week of E or C but the rarely do it with prop. add in the fact that the long esters compound over time and that im sure plays a big role as well
swellonemm
New member
nice oak. whats your view on this guy taking winstrol i say not for him he could achieve his goals with a test cycle. if hes trying to cut with a first real cycle i would suggest anavar being the lightest compound on the body. your view is greatly appreciated in this arguement.
THE-DET-OAK
IncreasedMyT @ ULV
i have yet to try winstrol-reason being is cause its bad on the joints-and cause ive found everyone to pretty much agree your not going to see the effects your looking for from it until your around 10% or below. bottomline if your much higher than that-test/diet/cardio is the best stack for cutting. and ya Anavar (var) is mild so why not
swellonemm
New member
thank you my knowlegeable friend finally someone with some experience...lol 
surfdog
New member
I still have not heard any info, besides someones option, as to why not to take whin and why to take Anavar (var). (besides sides, thanks to D)
Personally I have no idea about var, but am currently going to be taking whin in my cycle. As for esters I am doing long but have also done short before, since your asuming I have not done them.
I don't have a problem with someone suggesting one compound over another. But I do with telling someone what they should take. Espeically when they both used for similar goals.
Personally I have no idea about var, but am currently going to be taking whin in my cycle. As for esters I am doing long but have also done short before, since your asuming I have not done them.
I don't have a problem with someone suggesting one compound over another. But I do with telling someone what they should take. Espeically when they both used for similar goals.
swellonemm
New member
YOU SERIOUS. YOU WROTHE THIS IN AN OLD THREAD, THIS IS NOT CONSIDERED USING SHORT ESTERS, YOU HAVE NO EXPERIENCE WITH AAS USE. JUST SIT BACK AND LEARN A BIT.THANKS.
SURFDOG-At first I was doing 100ml e3d, about 5-6 hours later fever would start, only last fist night and not always, but always had redness. Other dudes taking it were fine and doing 2 or 3 times that. I only did that for 5 weeks. Then I took dbol 30ml a day and 50-75mls of prop e3d for 6 wks. I ended up giving the rest away and they didn't have any reactions like that either.
SURFDOG-At first I was doing 100ml e3d, about 5-6 hours later fever would start, only last fist night and not always, but always had redness. Other dudes taking it were fine and doing 2 or 3 times that. I only did that for 5 weeks. Then I took dbol 30ml a day and 50-75mls of prop e3d for 6 wks. I ended up giving the rest away and they didn't have any reactions like that either.
surfdog
New member
and whats your point? I didn't do a short ester lol that was test p hmmm what now not a short lol, or that my body didn't react well to the gear. and why can't you give me a reason for Anavar (var) over whin. Simple question for someone who knows all. everything I read about Anavar (var) is a fat burner, and hardner. Whin has similar effects also.
As for how I was taking e3d that was before I knew any better, and was listening to "some guy in the gym" I didn't do everything the right way, which is how you learn to do things right.
Stop being a dushbag and answer the question, you have so much to say, but still nothing that supports your statments. Do you really think your way is the only way?
and isn't that what "I'm doing trying to learn" a real reason why "whin isn't for him" but "he should do Anavar (var) instead"
Last edited:
swellonemm
New member
anavar isnt a fat burner.. say you used clen and anavar in a cycle, anavar will sqeeeeeeze the fat out, and the clen wil burn that fat off, anavar is just to harden up or gain some strength, leaner muscle gain, Winstrol (winny) on the other hand really just makes you dry..... meaning, It hardens up the muscles and makes ALREADY lean people vascular. know what im sayin, Winstrol is much rougher on the body as well. Thats why i suggest the use of anavar.
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