After about two months I have been interested in steroids and researching them constantly. I' am Currently 6'1 and weigh about 210. I was thinking about taking winstrol for 1 cycle for getting cut. Can someone tell me what I use with this during my cycle and post cycle so I can keep my results?
Okay I'am new at this...
- Thread starter yankee44
- Start date
THE-DET-OAK
IncreasedMyT @ ULV
If you have done so much research-then why would you pick out an oral only cycle? Why would you use Winstrol (winny) on your first run? What is your bf%? How long have you been training? How old are you?
brolickant
New member
test have to be the first compound for a first cycle. how old are you? test can get you cut up with a decent diet and training. Winstrol (winny) is suggested for people at 12% BF and lower.
brolickant
New member
and trust me, you think you have done alot of research on AAS but you havnt. i thought i did also but i am on hear almost every day asking questions. 
for test you can start of with 200mg a week for 10 weeks total. for post cycle therapy (pct), you can take nolva at 40mg for the first 2 weeks and 20 for the remainding 2 wks. (pct starts 2 wks after last injection)
for test you can start of with 200mg a week for 10 weeks total. for post cycle therapy (pct), you can take nolva at 40mg for the first 2 weeks and 20 for the remainding 2 wks. (pct starts 2 wks after last injection)
Last edited:
lcgator
New member
You can cut on any steroid. You should wait until you get older IMO. Also get your diet right before starting anything.
lcgator
New member
He just made a type-o. Your right.
THE-DET-OAK
IncreasedMyT @ ULV
Everyone who is over 40 says they would love to have the natural test levels of someone your age. When you start taking steroids at such a young age you are increasing the chance that your balls will not produce as much test as they should-for as long. I am 29- I would hate to have to inject my self with test every week for the rest of my life, because my body wont produce enough. Maybe one day it will come to that, but hopefully that will be 10 or more years from now.
Also by doing steroids at an age younger than 25 can stunt your growth because your growth plates have not closed yet.
Also by doing steroids at an age younger than 25 can stunt your growth because your growth plates have not closed yet.
brolickant
New member
your right, sorry its 40mg...sorry for the error i thought i corrected it
Bro you're 19. Why would you want to use steroids when you can make awsome gains natural? Keep working out for another 2-5 years and read up more on AAS at the same time. Then you we can work on a cycle, but not now.
THE-DET-OAK
IncreasedMyT @ ULV
The key word in your statement is typically. No journal here-just learning from some people who have been there done that. I just saw a thread the other day that it is possible to grow past 25. either way he is 19-Are you saying its a good Idea to shut your balls down at that age? Why cause you did it? I will do some research now-I had previously thought it was useless because everyone generally would agree that he is too young-but give me a lil to get some more real info.
THE-DET-OAK
IncreasedMyT @ ULV
Anatomy & Development
Epiphyseal plates are present in growing bones. For example, they are found in children and are present through puberty and early adulthood.[1] These plates are located at one or both ends of bones between the epiphysis (end) and the diaphysis (shaft) of long bones. Growth occurs towards the diaphysis or shaft of the long bone.[2] In most adults, the epiphyseal plates ossify by the mid twenties and growth arrests.[1]
thats from wikepedia.
THE-DET-OAK
IncreasedMyT @ ULV
What Causes Growth Plate Injuries?
Growth plate injuries happen for many reasons. Most occur after a sudden accident, such as falling or being hit hard on the leg. People who sometimes get injuries from overuse include:
Gymnasts who practices for hours on the uneven bars
Long-distance runners
Baseball pitchers perfecting their curve balls.
The top reasons for growth plate injuries are:
Falling down
Competitive sports (like football)
Recreational activities.
Other reasons for growth plate injuries are:
Child abuse
Injury from extreme cold (for example, frostbite)
Radiation (used to treat certain cancers)
Medications (for example, steroids)
Neurological disorders that cause people to lose their balance and fall
Some inherited disorders
Bone infections
Metabolic disease, such as kidney failure and hormone disorders.
Thats from the national institute of health and human services.
Growth plate injuries happen for many reasons. Most occur after a sudden accident, such as falling or being hit hard on the leg. People who sometimes get injuries from overuse include:
Gymnasts who practices for hours on the uneven bars
Long-distance runners
Baseball pitchers perfecting their curve balls.
The top reasons for growth plate injuries are:
Falling down
Competitive sports (like football)
Recreational activities.
Other reasons for growth plate injuries are:
Child abuse
Injury from extreme cold (for example, frostbite)
Radiation (used to treat certain cancers)
Medications (for example, steroids)
Neurological disorders that cause people to lose their balance and fall
Some inherited disorders
Bone infections
Metabolic disease, such as kidney failure and hormone disorders.
Thats from the national institute of health and human services.
THE-DET-OAK
IncreasedMyT @ ULV
How Well Do Children Grow After a Growth Plate Injury?
Most growth plate fractures get better and do not cause any lasting problems. Occasionally, the bone stops growing and ends up shorter than the other limb. For example, a fractured leg might end up shorter than the other leg. Or, if only part of the growth plate is injured, the limb can become crooked when only part of the bone keeps growing.
Lasting problems are most common with injuries to the knee.
same site as the previous.
Most growth plate fractures get better and do not cause any lasting problems. Occasionally, the bone stops growing and ends up shorter than the other limb. For example, a fractured leg might end up shorter than the other leg. Or, if only part of the growth plate is injured, the limb can become crooked when only part of the bone keeps growing.
Lasting problems are most common with injuries to the knee.
same site as the previous.
brolickant
New member
i agree, but 19 is still kinda young, their are alot of guys out their that took AAS 16 to even 25 thats on TRT.
THE-DET-OAK
IncreasedMyT @ ULV
So let me get this straight
1: He is not done growing?
2:Taking steroids increase the risk of injuring those plates
3:taking test without working out for a prolonged period of time-will increase the chance of hurting yourself because of the rapid strength increases.
4:injuring or fracturing a plate can cause it to stop growing-although your other bones will keep growing so one limb will be shorter than the rest if injured.
5:He may or may not be able to produce as much sperm later-because test is the only hormone that has to be involved in the sperm production process. Since he is jeopardizing his ability produce high levels of test later in life.
6:At age 19 he is old enough to make a decision that he may or may not want kids later?
Now that we have went over just a few of the negative consequences of injecting test at such a young age-lets review the benefits.
anyone?
1: He is not done growing?
2:Taking steroids increase the risk of injuring those plates
3:taking test without working out for a prolonged period of time-will increase the chance of hurting yourself because of the rapid strength increases.
4:injuring or fracturing a plate can cause it to stop growing-although your other bones will keep growing so one limb will be shorter than the rest if injured.
5:He may or may not be able to produce as much sperm later-because test is the only hormone that has to be involved in the sperm production process. Since he is jeopardizing his ability produce high levels of test later in life.
6:At age 19 he is old enough to make a decision that he may or may not want kids later?
Now that we have went over just a few of the negative consequences of injecting test at such a young age-lets review the benefits.
anyone?
THE-DET-OAK
IncreasedMyT @ ULV
Moshe Phillip1, 3 , Gila Maor2, Sara Assa1, Aviva Silbergeld1 and Yael Segev3
(1) The Felsenstein Medical Research Center, Institute for Endocrinology and Diabetes, National Center for Childhood Diabetes, Schneider Children’s Medical Center, Petach Tikva, Israel
(2) Department of Morphology Science, The Rappaport Faculty of Medicine, Technion, Haifa, Israel
(3) Molecular Endocrine Laboratory, Ben-Gurion University of the Negev, Beer-Sheva, Israel
Received: 27 February 2001 Revised: 2 June 2001 Accepted: 9 July 2001
Abstract Puberty is associated with an increased in the plasma concentration of sex steroids, growth hormone (GH), and insulin-like growth factor-1 (IGF-1). Gonadal steroid hormones are important for the normal pubertal growth spurt and skeletal growth. The mechanism by which gonadal steroids induces skeletal growth is still not fully understood. To study the GH-independent effect of testosterone on growth, we investigated the effect of testosterone injections on the tibial epiphyseal growth plate (EGP) in an in vivo model of hypophysectomized and castrated male rats. Four groups (six animals each) of 28-d-old male rats were studied. Groups A, B, and C were hypophysectomized and castrated and received 500 µg/(kg·d) of hydrocortisone and 15 µg/(kg·d) of levothyroxine sodium. Groups A and B were also treated with daily sc injections of 10 µg of testosterone/100 g of body wt, and 100 µg of testosterone/100 g of body wt, respectively, for 7 d. Group C was injected with vehicle alone. Group D were intact animals injected with saline (controls). Animals were sacrificed on 8 d. As expected, serum GH levels were found to be very low (1.13±0.1 ng/mL) in the hypophysectomized animals (group C, hypopit), and testosterone treatment did not change them significantly. Serum IGF-1 decreased from 502.9± 13 ng/mL in group D to 167±41.4 ng/mL in group C (p<0.001). Testosterone therapy had no stimulatory effect on serum IGF-1 levels in the hypopit + low-dose group (A) (220±94.8 ng/mL) and had an inhibitory effect in the hypopit + high-dose group (B) (39.3±17.5). Histomorphometric determinations demonstrated an EGF width of 472.3±39 µm in the intact animals but only 336.9±1.6 µm in the hypopit group (C) (p<0.01). High-dose testosterone treatment (group B) significantly increased the EGP width (to 438.8±27.8), (p<0.001), whereas low-dose testosterone (group A) did not. Immunohistochemistry studies revealed that the levels of IGF-1 in the EGP of the control animals were almost negligible and that testosterone did not change them. However, testosterone increased in a dose-dependent manner the abundance of IGF-1 receptor EGP. We conclude that testosterone has a direct, local, GH-independent effect on the EGP growth and IGF-1 receptor abundance.
Key Words Testosterone - IGF-1 - epiphyseal growth plate
Moshe Phillip (Director)
Email: Mosheph@post.tau.ac.il
Fulltext Preview (Small, Large)
If I am reading that right-the group that recieved the high dose of test-the test had an inhibitory effect on gh.
(1) The Felsenstein Medical Research Center, Institute for Endocrinology and Diabetes, National Center for Childhood Diabetes, Schneider Children’s Medical Center, Petach Tikva, Israel
(2) Department of Morphology Science, The Rappaport Faculty of Medicine, Technion, Haifa, Israel
(3) Molecular Endocrine Laboratory, Ben-Gurion University of the Negev, Beer-Sheva, Israel
Received: 27 February 2001 Revised: 2 June 2001 Accepted: 9 July 2001
Abstract Puberty is associated with an increased in the plasma concentration of sex steroids, growth hormone (GH), and insulin-like growth factor-1 (IGF-1). Gonadal steroid hormones are important for the normal pubertal growth spurt and skeletal growth. The mechanism by which gonadal steroids induces skeletal growth is still not fully understood. To study the GH-independent effect of testosterone on growth, we investigated the effect of testosterone injections on the tibial epiphyseal growth plate (EGP) in an in vivo model of hypophysectomized and castrated male rats. Four groups (six animals each) of 28-d-old male rats were studied. Groups A, B, and C were hypophysectomized and castrated and received 500 µg/(kg·d) of hydrocortisone and 15 µg/(kg·d) of levothyroxine sodium. Groups A and B were also treated with daily sc injections of 10 µg of testosterone/100 g of body wt, and 100 µg of testosterone/100 g of body wt, respectively, for 7 d. Group C was injected with vehicle alone. Group D were intact animals injected with saline (controls). Animals were sacrificed on 8 d. As expected, serum GH levels were found to be very low (1.13±0.1 ng/mL) in the hypophysectomized animals (group C, hypopit), and testosterone treatment did not change them significantly. Serum IGF-1 decreased from 502.9± 13 ng/mL in group D to 167±41.4 ng/mL in group C (p<0.001). Testosterone therapy had no stimulatory effect on serum IGF-1 levels in the hypopit + low-dose group (A) (220±94.8 ng/mL) and had an inhibitory effect in the hypopit + high-dose group (B) (39.3±17.5). Histomorphometric determinations demonstrated an EGF width of 472.3±39 µm in the intact animals but only 336.9±1.6 µm in the hypopit group (C) (p<0.01). High-dose testosterone treatment (group B) significantly increased the EGP width (to 438.8±27.8), (p<0.001), whereas low-dose testosterone (group A) did not. Immunohistochemistry studies revealed that the levels of IGF-1 in the EGP of the control animals were almost negligible and that testosterone did not change them. However, testosterone increased in a dose-dependent manner the abundance of IGF-1 receptor EGP. We conclude that testosterone has a direct, local, GH-independent effect on the EGP growth and IGF-1 receptor abundance.
Key Words Testosterone - IGF-1 - epiphyseal growth plate
Moshe Phillip (Director)
Email: Mosheph@post.tau.ac.il
Fulltext Preview (Small, Large)
If I am reading that right-the group that recieved the high dose of test-the test had an inhibitory effect on gh.
THE-DET-OAK
IncreasedMyT @ ULV
My point is to prove that him calling bs is bs.
StoneColdNTO
Administrator
You are wrong.......110% wrong.
and trust me, you think you have done alot of research on AAS but you havnt. i thought i did also but i am on hear almost every day asking questions.
for test you can start of with 200mg a week for 10 weeks total. for post cycle therapy (pct), you can take nolva at 40mg for the first 2 weeks and 20 for the remainding 2 wks. (post cycle therapy (pct) starts 2 wks after last injection)
for a 210lb guy i would start with 400mg of test cypionate (longer ester, you only have to inject once per week). and you should take 1mg of arimidex every day to help stop water retention and gyno. take Human Chorionic Gonadotropin (HCG) for post cycle therapy (pct) to help with testicular atrophy
what you said was a crappy first cycle. mine is much better balanced...
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