ulter said:
"how are you so sure that Human Chorionic Gonadotropin (HCG) is going to give someone gyno? if someone is running test at 1g a week and dbol at 35mg for the first 4 weeks, its not going to be the Human Chorionic Gonadotropin (HCG) that gives them gyno."
They add the Human Chorionic Gonadotropin (HCG) for the first time and get a flare up that is gone upon cessation of the HCG.
The rest of what you posted shows you're a little confused in this area.
1. Human Chorionic Gonadotropin (HCG) doesn't make your testes produce test on cycle. So no that's not why you shouldn't use it.
2. People DO take just Human Chorionic Gonadotropin (HCG) to help raise their test levels. But no you won't ever make your body produce enough to cycle from.
3. You can not take a little arimidex and think that your E levels won't be elevated. Arimidex only lowers E levels in NON CYCLING men by 59%. So cycling men are only a fraction of that. There is plenty of E left over to cause gyno. Especially when you're spiking it every few days.
4. There are no answer to when gyno may become a problem but gyno is only one of the evils of estrogens.
What is the "flare up" you are talking about? I dont understand what you are trying to say with that.
1. The only way i can think of Human Chorionic Gonadotropin (HCG) causing gyno is if it causes your testes to produce test, then that test aromatizes to estrogen and you get gyno. If it doesn't even make them produce test then how can it possibly cause gyno?
2. I was being sarcastic with that remark. My point was if gyno from 500mg of test a week is preventable, than gyno from Human Chorionic Gonadotropin (HCG) is also. Since you can't produce enough natural test with Human Chorionic Gonadotropin (HCG) to do a cycle then you probably dont have any greater risk of getting gyno from Human Chorionic Gonadotropin (HCG) than a cycle, and most people will agree if you have anti e's then you wont get gyno (this is dose dependant).
Also, i am not saying Human Chorionic Gonadotropin (HCG) is taken to raise test levels like a natural test booster, although if someone were taking Human Chorionic Gonadotropin (HCG) and not on a cycle this would probably happen because Human Chorionic Gonadotropin (HCG) mimicks lh, and that itself will cause the testes to produce more test, correct? WHen you are on a cycle, your nuts shrink because your hpta is shut down at the pituatary, and therefore no lh is released to stimulate the testes. By providing your body with Human Chorionic Gonadotropin (HCG) (which has the same effects in the testes as lh), then they don't atrophy, and when you stop your cycle lh levels rise, and you are no longer shut down. After a cycle lh levels rise pretty quickley, the reason you stay shut down is because the testes have 1) atrophied and can't produce test despite the lh and 2) are desensitized to lh, correct?
3. THis goes back to my original point. If you are using an anti e at a dose where you dont get gyno from a heavy Anabolic Androgenic Steroids (AAS) cycle, then i dont think taking Human Chorionic Gonadotropin (HCG) is going to cause a sharp rise in test that will aromatize and cause you to have gyno. Remember, the dose of anti e your taking should prevent you from getting gyno at a large dose of aas, so it should be sufficient for any test put into the system by the testes if Human Chorionic Gonadotropin (HCG) is taken (personally i dont think there will be much test released on a cycle so i still have no idea how it will give you gyno).
