Tren . What do you guys personally notice/get from it

[Milton]

I don't know if it will work for everyone but this is how I fixed my insomnia from tren. I bought a window unit air conditioner. Some of you rich fellows could probably just turn the thermostat down to 60. That and I would stay well hydrated before bed. Even still, my morning pee would be honey colored. Before these charges it would be much darker. I think something about hydration and sweating were contributing factors. Give it a shot sometime.

Lets keep it real as we know who is here at all times.

 

[Milton]

But you and Todd are the same person, that's pretty clear to me.

Lets keep the drama to the PM's we dont need another round of back and forths again. Please.

 

[Milton]

Onk and Schredder are hardly newbs. They are both well respected members around here.

Its best for Det-Oak to PM DPR as we dont need more drama here after last week.

 

[bigben66]

I'll say no more on the matter, and I don't need PM's either.

I say what I see... End of.

 

[Dreaded Pirate Roberts]

That it really shuts you down hardcore, more than anything else.

I've run it heavy and for a long time and sure it shuts you down but once your shut down your shut down. The strength and positives so out weighed the negitivety. The chalanges I faced was the need to keep my kidneys hydrated.

 

[THE-DET-OAK]

I agree on shut down, my point is that things like DHT and estrogens don't bounce right back. Tren is so suppressive its actually more suppressive than estrogen. Which is hard to do.

So we have shut down, then we have getting derivatives like DHT going that don't come back right away because there is no calling for it. You have now reached homeostasis with Tren so your body has no need for conversions. This affects thyroid, adrenals etc

I see this phenomena in men who use anastrozole too. After a while they wont need as much, because their body just stops aromatizing.

You can see this same affect in men who took propecia for long periods as well.

Tren has such a strong affinity it can stick to E receptors as well.

 

[Dreaded Pirate Roberts]

Great info as always from a mutual friend and it cause me to start reading.

A random thought on the subject and Im just spit ballin. Could a GnRH antagonist be of any use during a cycle that includes Tren? LH and FSH are already shut down. If one was to include a GnRH antagonist alongside HCG DURING the cycle would it make the following Nolvadex/Clomid treatment more effetive if Tren does in fact downregulate GnRH receptors?

Thoughts?

Igf1-lr3 is what you seek.

 

[Black Beard]

Boy..what happened to this place....

 

[Dreaded Pirate Roberts]

Esteogen will bounce back depending on the compound used. That is why I love letro the bounce back ads in recovery.

 

[THE-DET-OAK]

Case in point:

There's why you are sleeping; the Propecia. DHT is the most CNS stimulating form of testosterone....your adrenaline levels are taking a dump and so you have less awareness too.
Estrogen low is also playing a role, but I recommend you stop fucking with your DHT.
aka...Get off the damn Propecia bro ^_^

Just my two cents.

http://www.steroidology.com/forum/a...ptoms-estrogen-being-too-low.html#post3442602

 

[Southernfish]

Igf1-lr3 is what you seek.

Ive ran LR3 a number of times. It is definitely not used for this purpose. It one relied on it for this purpose he would be in trouble.

 

[Southernfish]

I see this phenomena in men who use anastrozole too. After a while they wont need as much, because their body just stops aromatizing.

Do all men using Anastrozole experience this?

 

[THE-DET-OAK]

Do all men using Anastrozole experience this?

No but a lot do after a year or so. Some can probably be attributed to decrease fat but I've seen their E crash and not want to come back for 6 months even after discontinuing.

Obviously not all cases are as extreme.

 

[Samson4]

Lets keep it real as we know who is here at all times.

What was "not keeping it real" about my post?

 

[THE-DET-OAK]

just broke a thousands hits, noice....... my milk shake brings all the boys to the yard

It gets the people GOING!!

 

[halfwit]

Uhhhhhhhhhhh, I'm under the impression that 19-nors have zero antigonadotropic activity on the receptors themselves. This is why birth control pills have been around forever, as the use such derivatives to suppress LH and FSH in women, which are far more difficult to completely suppress - but only temporarily.

Hmm, as far as the OP topic goes: I absolutely love and hate trenbolone. I take it when I am able to forget just how shitty it makes me feel after prolonged use. It's liver toxic, kidney toxic, and messes with my brain in ways I don't care for - but it's INCREDIBLY good at what it does.

If there were a better way to go about building my body with something that was less harsh, but provided the same results - I'd be on it in a heart beat.

Yes, I've done low test, high test, DA use, short esters, long esters, carb starvation, carb surplus, and pretty much about any variable that is supposed to mitigate tren's nasty sides.

But I'll keep pinning it until I'm happy.

 

[THE-DET-OAK]

Again suppression, and suppression of derivate ratios are two different things. So in the sense your talking about as far as whether the HPGA will make testosterone? Correct as for Tren wreaking havoc on most of the receptors it can find suppressing aromatase and DHT conversion, I have to disagree.

 

[THE-DET-OAK]

When you get time just search the board for threads of people not recovering from Tren and then look at their lab work. Its very easy to see.

 

[halfwit]

Again suppression, and suppression of derivate ratios are two different things. So in the sense your talking about as far as whether the HPGA will make testosterone? Correct as for Tren wreaking havoc on most of the receptors it can find suppressing aromatase and DHT conversion, I have to disagree.

But by what method of action would this entail? While I agree that it has a far greater binding affinity at the receptor, I am not familiar with any antiandrogenic properties inherent with trenbolone (or nandrolone) by acting upon the metabolic pathways responsible for cleaving of testosterone to its derivatives.

To my understanding, the chief issue with this particular class of hormones is their progestingenic capacity, which does (oddly) lower estradiol, while increasing prolactin. As the feedback loop is open, gnrh inhibition shouldn't be of any consequence, or are you referring to once the compound has been ceased?

I can see lasting effects from such an impact on the HPTA; especially if the individual neglected proper estradiol control with an aromatizing agent, but I would imagine that suppression at the hypothalamus and/or pituitary would be far more prominent, no?

 

[THE-DET-OAK]

That is a common misconception IMHO. Not talking about nandrolone honestly it and Tren are quite different. For instance when men take testosterone their DHT conversion rates go up, when they take Tren it goes way down because they do not need it. Tren replaces DHT, there is no need for DHT anymore.

Same thing with Estrogen. This same exact thing happens for men who use propecia go to their board and look at the labs.

Estradiol and DHT are made in the testes, this has nothing to do with GRNRH or the other rungs down the ladder.

Making these derivatives takes a lot longer to come back than simply getting some T produced, not to mention we don't have anything to stimulate these processes. As a matter of fact your body is working against you because your TT levels are low and it is trying to preserve all the T it can.