i disagree ....i think with an sd clone and tren(ph)clone, an otc pct like the one you outlined is not the safest way to go about things
most definately it can work, and will for some, but to say a serm is not warranted or at least within arm's reach is a little 'off' for my liking
why dink around with a bunch of different otc supps, one toremifene will work 100%, is cheaper, and more effective
for a hdrol clone sure, maybe even an sd or tren clone solo, but the combo the op wants to run is extremely suppressive, not to mention he has no on cycle test/dht base, thus creating a horrible imbalance in his cycle and a rapid and severe shutdown of the hpta
to the op, good luck with whatever you plan to run
***never tamox/nolva with a nor compound, in this case the tren clone.....clomid or torem
Well first off all my friend Forma-stanzol is not just some supplement lol.. Some of the ingredents were drugs long before clomid and nolva was around.. Forma-stanzol is a suicide aromatase inhibitor (letheron tm) as well as serms and more all in one.
A serm without a suicide aromatase inhibitor after a beastdrol cycle is more likely to cause rebound. During beastdrol/superdrol more often then not estrogen will be low (because as you said its a dht) and when coming off a rebound accrues.....
Anyone and everyone who knows how to read a study and research knows good and well the anabolic
although "formastan" its self has nothing in common at all with nolva or clomid, forma-stanzol does. Nolva and clomid are serms and they block estrogen at the ER thus stopping it from having any effects on the ER.
However when on nolvadex or clomid the level of estrogen in your body does not go down but rather it goes up!!!!. You see, Nolvadex only blocks estrogen that's already in your body but it does not in anyway effect test from converting to estrogen. In fact by stopping the estrogen from entering the ER it causes your body to create both more testosterone AND ESTROGEN :biggrin::biggrin: let no one ever forget this. Your bodies level of estrogen goes up well taking nolvadex or clomid not down and anyone who has read a study or two and has been around a wile knows this. This is why it is very very very common for nolvadex and or clomid to cause what is known as "rebound gyno" if you use these product for pct with out a Aromatase inhibitor (AI) or even better a "suicide aromatase inhibitor" (which is what forma-stanzol is)
Estrogen going up during pct is not a good thing .
Now as you can see formastane its self and nolvadex/clomid are worlds apart. From here on throughout my explanation I will be speaking only about forma-stanzol as I feel it is the far superior compound because it has both formastane (suicide aromatase inhibition as well as progesterone reducing effects) and phytoserms (
Phytoserm - Wikipedia, the free encyclopedia) effects. On top of this it also has many other necessary effects for any prospective steroids user.
One Forma-stanzol's many characteristics is whats know as a "suicide aromatase inhibitor" of aromatase. This means that Forma-stanzol binds to the aromatase enzyme in a permanent and irreversible manner, rendering it inactive.
The result of this is an eventual diminishment of aromatase enzyme in the body and a concurrent reduction in estrogen levels. A corresponding increase in testosterone production is usually experienced as well
It is important to note here that this deactivation of aromatase enzymes by forma-stanzol does not mean that your body becomes permanently deficient in the ability to synthesize estrogen. Your body will react to the deficiency of enzyme by producing more enzyme to replace that which has been deactivated. Therefore, when you stop taking Forma-stanzol your aromatase enzyme level will quickly catch up to normal and full estrogen production will resume.
Now Another important attribute to forma-stanzol is of course its phytoserm effects. Serm/phytoserm effects are important for pct because of there binding to the estrogen receptors, thus inhibiting estrogenic activity only at the ER. This causes a increase to LH & FSH levels, which in turn stimulates testosterone production. The important thing to remember here is although both nolvadex and clomid will do this
they do it at a price! not only does the level of estrogen keep going up well taking them (unlike with forma-stanzol) but they can also be harmful in many other ways ( read post 6 in this thread to learn more about this. http://www.****************/forum/anabolic-steroids/taking-anabolic-steroids-101-a-642856.html). This is why phytoserm's "medically and clinically" excepted natural serms are better. Combined with other compounds like the ones in forma-stanzol they are a much more effect form of pct or on cycle estrogen and progesterone control.
Furthermore forma-stanzols 7,8 Benzoflavone a neuro-active flavone has the ability to pass the blood brain barrier and block the suppression of GnRH release through modulation of the GABAergic receptor complex.7,8 Benzoflavone also has a positive effects on libido due to its aphrodisiac and anxiolytic (anxiety-relieving) effect having natural anti-anxiety properties, 7,8-benzoflavone my help improve general self-confidence and well being. But Forma-stansols posative effects on libido dosn't end there. As any well versed steroid user knows lowering progesterone can also have a very positive and profound effects on sex drive.
Forma-stanzol unlike any other Aromatase inhibitor (AI) or serm also has anabolic effects and coverts to a anabolic at a "dose dependent rate". In other words when used at the higher end of dosing ( 10 pumps twice a day) after a week it starts to covert somewhat to a anabolic compound and adds gains to your cycle.
Still when used at the lower end or pct stile dosing protocol ( 5 pumps twice a day) there is no worries about suppression because its anabolic conversion is again " dose dependent" and only happens at higher doses taken for longer periods of time. How Amazing is that? I dont know any Ai's out there that can clam this nor do I know one single Aromatase inhibitor (AI) that also lowers progesterone too!
Because of the formatane and now added compounds in forma-stanzol Its anabolic/androgen effects are similar to that of the steroid primobolan Depot ( but only when used at higher doses for longer periods of time).even at the lower dosing It increases IGF-1 levels by an amazing 26%,and increases HPTA activity and testicular activity similar to a combination of hcg and Clomid!
All of this is backed up by " human" studies. Yes Real human studies don by well known Universities and agencies. Because for the longest time Lentaron I.M. Depot® was a proscription drug . This was not a drug that got scrapped because it did not work or because other drugs worked better. No this drug lost favor because many years ago the only way to use the drug was through injections. But because of the advancements in Trans dermal delivery Lentaron I.M. Depot® is back. With the help of NTBM and MRsupps.com its more powerful then ever.
forma-stanzol is a synergistic blend of supporting components making forma-stanzol a Highbred on cycle estrogen/progesterone control and pct drug.
Aromatase inhibition: 4-hydroxyandrostenedione (4-OHA, CGP 32349) in advanced prostatic cancer.
Comparison of the pharmacokinetics and pharmacodyn... [Cancer Chemother Pharmacol. 1990] - PubMed result
Biochemistry and function of sterols - Google Books
Pharmacokinetics of 4-hydroxyandrostenedione in ma... [J Steroid Biochem Mol Biol. 1993] - PubMed result
In each of these studies we can see that despite that fact that 4-hydroxyandrostenedione has also ( in some studies) shown to be a "very weak prohormone" to the anabolic steroid 4-hydroxytestosterone, as an aromatase inhibitor it also possesses notable testosterone stimulating properties. The use of 4-hydroxy 4-androstenedione gradually increase androgen levels while simultaneously decreasing estrogen and dihydrotestosterone (DHT). Natural androgen production is increased by the stimulation of the hypothalamic pituitary axis via an increase in luteinizing hormone (LH) levels coupled by the direct stimulation of testosterone by 4-hydroxy 4-androstenedione. Luteinizing hormone (LH) is responsible for the production of total serum testosterone and testicular function. Formastane gives a gradual decrease in estrogen blood concentration which produces a signal of the hypothalamic pituitary ( again despite its very weak conversion to 4-hydroxytestosterone).
Whatever "weak conversion activity" it may have is more than compensated for by its ability to drop estrogen levels. This action of course reduces the suppressive signal estrogen sends to your brain.
In all of these studies Formastane was shown to suppress estrogen concentrations yet dos not suppress gonadotropins .
Let me further help you understand the difference and why one is good for pct (suicide aromatase inhibitors) and the other is not.
To understand why forma-stanzol may be useful while regular ai's (like Letro and A-dex) are not we'll need to first understand the differences between
the two. suicide aromatase inhibitors are actually steroidal compounds, while regular Ai's (like Letro and A-dex) are non-steroidal drugs ( no I don't mean STEROID LIKE YOU ARE HOPING LMOA)
Of course, there are some similarities between the two types of AIs...both
suicidal and non suicidal AIs mimic normal substrates (essentially androgens), allowing them to compete with the substrate for access to the binding site on the aromatase enzyme. After this binding, the next step is where things differ greatly for the two different types of AI's. In the case of a suicide aromatase inhibitors the noncompetitive inhibitor will bind, and the enzyme initiates a sequence of hydroxylation ( hence the name 4-hydroxy given to formastane) ; this hydroxylation produces an unbreakable covalent bond between the inhibitor and the enzyme protein.
Now, enzyme activity is permanently blocked; even if all unattached inhibitor is removed. Aromatase enzyme activity can only be restored by new enzyme synthesis.
Now, on the other hand, most all other Aromatase inhibitor (AI) are competitive inhibitors, reversibly bind to the active enzyme site, and one of two things can happen: 1.) either no enzyme activity is triggered or 2.) the enzyme is somehow triggered without effect. Thus essentially they can actually disassociate from the binding site, eventually allowing renewed competition between the inhibitor and the substrate for binding to the site. This means that the effectiveness of competitive aromatase inhibitors depends on the relative concentrations and affinities of both the inhibitor and the substrate, while this is not so for noncompetitive inhibitors. Forma-stanzol is a suicide aromatase inhibitor meaning that once it has done its job, and deactivated the aromatase enzyme, we don't need it anymore! Starting to understand now bro? Other Ai's need to remain present to continue their effects and this is why they are not the best choice for use during pct but forma-stanzol is..